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hydrops fetalis/upala
Veza se sprema u međuspremnik
Stranica 1 iz 36 rezultatima
Hydrops fetalis from B19 parvovirus infection.
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Human B19 parvovirus infection may be associated with hydrops fetalis and perinatal loss. This report documents a case of unexplained elevation of maternal serum alpha-fetoprotein with normal amniotic fluid alpha-fetoprotein before hydrops and in the absence of placental inflammatory reaction. More
Human parvovirus B19: a new emerging pathogen of inflammatory cardiomyopathy.
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The human parvovirus B19 (PVB19), an erythrovirus causing diverse clinical manifestations ranging from asymptomatic or mild to more severe outcomes such as hydrops fetalis, is the only known human pathogenic parvovirus so far. Although enteroviruses have long been considered the most common cause of
Parvovirus B19: a new emerging pathogenic agent of inflammatory cardiomyopathy.
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The human parvovirus B19 (PVB19), an erythrovirus causing diverse clinical manifestations ranging from asymptomatic or mild to more severe outcomes such as hydrops fetalis, is the only currently known human pathogenic parvovirus. Recently, PVB19 has been identified as a causative agent of pediatric
Fetal hydrops associated with spontaneous premature closure of ductus arteriosus.
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A 36-year-old woman presenting with fetal growth restriction in the 25th week of gestation was referred to Kobe University Hospital where hydrops fetalis was detected. A stillborn fetus, 2012 g in weight and 40 cm in height, was delivered in the 33rd week of gestation. The mother had no past history
Cardiac abnormalities associated with hydrops fetalis.
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Cardiovascular abnormalities are associated with hydrops fetalis in 26% of cases reported in the modern era. These include not only structural abnormalities, but also cardiac arrhythmias, failure, tumors, myopathy, infection, inflammation, infarction, and arterial calcification. Cardiac structural
Mesalazine as a cause of fetal anemia and hydrops fetalis: A case report.
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BACKGROUND
Mesalazine and its prodrug sulfasalazine are both used for inflammatory bowel disease. Sulfasalazine has been associated with hematological side-effects such as aplastic and hemolytic anemia in patients, but also in fetuses after intrauterine exposure. To our knowledge, we describe the
A 6-year-old girl diagnosed with mevalonate kinase deficiency who had hydrops fetalis and neonatal-onset cholestasis.
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We experienced a 6-year-old girl diagnosed with mevalonate kinase deficiency (MKD) who had cholestasis, anemia, and elevated inflammatory markers in neonatal period. She was admitted to our hospital because of fever and elevated inflammatory markers at 5 years 11months of age. Without using
Chronic histiocytic intervillositis with cytomegalovirus placentitis in a case of hydrops fetalis.
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Chronic histiocytic intervillositis (CHI) is an infrequent inflammatory placental disorder associated with unfavorable pregnancy outcomes and a high rate of recurrence. This disorder is thought to reflect a maternal delayed hypersensitivity response to fetal antigen(s) in placental tissue. We report
Congenital heart block in neonatal lupus: the pediatric cardiologist's perspective.
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METHODS
Congenital heart block (CHB) in the absence of major structural abnormalities is associated with maternal antibodies to Ro (SS-A) and La (SS-B). CHB is most commonly diagnosed between 18 and 24 wk of gestation, and may be first, second or third degree (complete). Mortality approaches
Pulmonary interstitial glycogenosis in a patient ultimately diagnosed with Noonan syndrome.
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We describe an infant prenatally diagnosed with hydrops fetalis ultimately found to have Noonan syndrome (NS). Prior to genetic confirmation of diagnosis, lung biopsy was performed which revealed widespread pulmonary interstitial glycogenosis (PIG), abnormal alveolarization, and mild inflammation.
Isolated congenital pleural effusion in two neonates.
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Congenital isolated pleural effusion, a non-specific accumulation of fluid in the pleural space, is an uncommon anomaly which can be associated with structural malformations, inflammatory or iatrogenic problems, genetic syndromes or fetal hydrops. Here, we present two neonates with isolated
Inhibition of polymorphonuclear leukocyte chemotaxis by alpha-macrofetoprotein, an acute-phase reactant of the rat.
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alpha-Macrofetoprotein (alpha M FP) is a normal fetal plasma constituent in the rat, with very low plasma levels in the adult phase but rising sharply after injury. This fetal acute-phase protein is a strong inhibitor of inflammatory edema. Fetal inflammatory reactions show diminished exudation, but
Repeated paracentesis in a fetus with meconium peritonitis with massive ascites: a case report.
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Meconium peritonitis (MP) is defined as a sterile inflammatory reaction in the fetal abdomen that is seen in cases of intrauterine bowel perforation. Recently, there have been increasing numbers of fetuses with MP prenatally diagnosed by ultrasonography. Massive fetal ascites in MP may cause hydrops
[Systemic manifestations of Parvovirus B19 infections].
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OBJECTIVE
Parvovirus B19 (B19) causes many clinical disorders, of which the most common are erythema infectiosum, aplastic crisis complicating chronic hemolytic anemia, and hydrops fetalis. In young adults, the skin eruption caused by B19 is accompanied by polyarthritis and polyarthralgia in 60% of
Placental and fetal pathology in intrauterine viral infections.
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Intrauterine viral infections show a wide range of fetal involvement with variable fetal outcome. The histopathologic changes of the placenta during viral infection show a wide spectrum. The most active processes are proliferative with a variety of cells involved. Vascular changes, including
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