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At toxic levels, CsA also has the ability to cause renal damage and histological changes that can affect the function of a transplanted kidney. Some of the typical physiological effects of CsA include reduced glomerular filtration and changes in intrarenal hemodynamic function, which can start to
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Extensive experimental evidence confirms the role of oxidative stress as a major contributor to the pathogenesis of acute kidney injury (AKI). However, less information is available on the evolution of prooxidant-antioxidant parameters from early to end-phase renal function decline in humans. This
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