biliary atresia/protease

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Evaluation of matrix metalloproteinases and their endogenous tissue inhibitors in biliary atresia-associated liver fibrosis.

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OBJECTIVE Matrix metalloproteinases (MMPs) and their endogenous tissue inhibitors (TIMPs) are major proteases responsible for remodeling the liver tissue, but their roles in biliary atresia (BA)--associated liver fibrosis are not clear. METHODS A DNA microarray containing complementary DNA clones of

Alpha 1-antitrypsin deficiency (Pi SZ) and biliary atresia.

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We report an infant who presented at 2 days of age with conjugated hyperbilirubinemia. Serological, radiographic, and surgical investigations revealed the concurrence of alpha-1-antitrypsin deficiency, protease inhibitor type SZ, and extrahepatic biliary atresia.

Matrilysin (MMP-7) is a major matrix metalloproteinase upregulated in biliary atresia-associated liver fibrosis.

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Matrix metalloproteinases (MMPs) are the proteases responsible for tissue remodeling during liver fibrosis caused by various disorders including biliary atresia. However, information regarding the relative contribution of these proteases to liver fibrosis is still limited. We studied matrix

[alpha 1-Antitrypsin deficiency in early childhood (author's transl)].

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alpha 1-Antitrypsin inhibits various proteases and excessive proteolysis. If serum and tissue concentrations of this compound are low throughout longer periods of time due to deficient synthesis, - which is a dominantly inherited trait, - progressive pulmonary emphysema will develop in adults, and

Antiretroviral therapies in pregnancy: maternal, fetal and neonatal effects. Swiss HIV Cohort Study, the Swiss Collaborative HIV and Pregnancy Study, and the Swiss Neonatal HIV Study.

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BACKGROUND Therapies containing two reverse transcriptase inhibitors (RTI) with or without protease inhibitors are used with increasing frequency in pregnant HIV-infected women. OBJECTIVE To assess the safety of antiretroviral therapy in pregnant women and their newborns. METHODS All clinical events

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