calligonum/antifonjik

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Antifungal activity in the methanolic, aqueous and hexane extracts of Calligonum polygonoides.

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Calligonum polygonoides is locally called as Phog which belongs to the Polygonaceae family. It is traditionally used as an antifungal. The methanolic extract, hexane extract, ethyl acetate extract and aqueous extract were screened against Candida albicans and Aspergillus niger in seven

In vitro kinetics and inhibition of krait snake's venom acetylcholinesterase by Calligonum polygonoides extract in relation to the treatment of Alzheimer's disease.

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UNASSIGNED The objective of this study was to evaluate pharmacological effect of Calligonum polygonoides against Krait snake's venom acetylcholinesterase (AChE) and to extent it for the treatment of Alzheimer's disease. UNASSIGNED Acetylcholinesterase activity was measured using Ellman method with

Barks Essential Oil, Secondary Metabolites and Biological Activities of Four Organs of Tunisian Calligonum azel Maire.

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This study is the first to investigate the chemical composition of barks essential oil (EO), secondary metabolites and biological activities of the MeOH and infusions extracts of seeds, leaves, barks and roots of Calligonum azel Maire (Polygonaceae) harvested from Tunisian desert. The gas

Immunomodulation of RAW 264.7 murine macrophage functions and antioxidant activities of 11 plant extracts.

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A group of 11 medicinal plants, including Lavandula pubescens, Trigonella foenugricium, Salsola schweinforthi, Calligonum comosum, Silene succulenta, Silene villosa, Bogonvillea glabra, Cakile maritime, Gomphrene celesoids, Mirabilis jalaba, and Silene nocturna growing in Egypt, were extracted and

Evaluation of the potential cardioprotective activity of some Saudi plants against doxorubicin toxicity.

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Doxorubicin (DOX) is an anthracycline antibiotic widely used as a chemotherapeutic agent in the treatment of several tumours. However, its cardiac toxicity limits its use at maximum therapeutic doses. Most studies implicated increased oxidative stress as the major determinant of DOX cardiotoxicity.

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