cerebral hemorrhage/hypoxia

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[Hypoxemia in the acute stage of hypertensive intracerebral hemorrhage, with special reference to increased extravascular lung water].

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Respiratory responses to acute brain damage have been well documented. Severe hypoxemia of unknown origin has often been seen in the acute stage of intracerebral and subarachnoid hemorrhage. Neurogenic pulmonary edema is a particular concern in the neurosurgical intensive care setting. In this

Hypoxia-inducible factor-1alpha accumulation in the brain after experimental intracerebral hemorrhage.

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Hypoxia-inducible factor-1 (HIF-1), a transcription factor composed of HIF-1alpha and HIF-1beta protein subunits, has been implicated in cellular protection and cell death in cerebral ischemia. The extent to which HIF-1 plays a role in brain pathology during intracerebral hemorrhage (ICH) is

Anemic hypoxia in moderate intracerebral hemorrhage: the alterations of cerebral hemodynamics and brain metabolism.

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To determine the influence of anemic hypoxia on cerebral hemodynamics and brain metabolism during pathological conditions of the brain, moderate-sized intracerebral hemorrhage (ICH) was created in canines with and without preoperatively inducing chronic anemia. The changes in cerebral perfusion

Neuroprotective effects of bone marrow stem cells overexpressing glial cell line-derived neurotrophic factor on rats with intracerebral hemorrhage and neurons exposed to hypoxia/reoxygenation.

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BACKGROUND Intracerebral hemorrhage (ICH) represents at least 15% of all strokes in the Western population and a considerably higher proportion at 50% to 60% in the Oriental population. OBJECTIVE To investigate whether administration of bone marrow stem cells (BMSCs) overexpressing glial cell

Effects of recombinant adenovirus-mediated hypoxia-inducible factor-1alpha gene on proliferation and differentiation of endogenous neural stem cells in rats following intracerebral hemorrhage.

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OBJECTIVE To investigate the effects of adenovirus (Ad)-mediated hypoxia-inducible factor-1alpha (HIF-1α) gene on proliferation and differentiation of endogenous neural stem cells (NSCs) in rats following intracerebral hemorrhage (ICH) and the underlying mechanisms. METHODS A total of 120 specific

[Intracerebral hemorrhage and its sequelae in high risk newborn infants in relation to oxygen deficiency status].

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In a prospective study 325 risk newborn infants were examined by means of ultrasound scanning. Children with and without intracerebral haemorrhages were classified, according to pre-, intra-, and postnatal periods of oxygen privation. For the characterization of short-term hypoxia the blood-gas

Hyperleukocytosis Complicated by Intracerebral Hemorrhage and Spurious Hypoxemia: A Case Report and Literature Review.

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We present the management of a 15-year-old girl with acute myeloid leukemia who presented with massive hyperleukocytosis and neurological deficit due to intracerebral hemorrhage. Surgical intervention was considered but ultimately not undertaken because of the presence of massive hyperleukocytosis,

Marked anemic hypoxia deteriorates cerebral hemodynamics and brain metabolism during massive intracerebral hemorrhage.

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The present study was undertaken to investigate the influence of imposed anemic hypoxia on cerebral hemodynamics and metabolism in a condition of massive ICH. Two groups of eight dogs, with a target hemoglobin concentration of 12 g/dl in nonanemic and 6 g/dl in anemic group, were included. Before

[Relationship between hypoxemia and urinary catecholamine excretion in subarachnoid hemorrhage and hypertensive intracerebral hemorrhage].

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[Correction of cerebral hypoxia by intracarotid ATP infusions in experimental intracerebral hemorrhage].

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[Pathogenetically based correction of cerebral circulatory hypoxia in the acute period of experimental intracerebral hemorrhage].

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ISCHEMIC ANOXIA IN HYPERTENSIVE INTRACEREBRAL HEMORRHAGE.

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[Pathogenesis of experimental intracerebral hemorrhages in the presence of hypertension in light of microcirculatory studies].

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The pathogenesis of early stages of cerebral hemorrhages was studied in albino rats with hypertension induced by an acoustic irritant in conjunction with hypoxia. Such hemorrhages developed during fibrinolysis activation and autoregulation failure of brain circulation marked by dilatation of

[Pneumothorax in the background of cerebral hemorrhage in premature infants].

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362 preterm newborns with birth weights ranging between 1 000 and 2 000 g were admitted to NICU of Kinderklinik Städtische Kliniken Kassel during Jan. 1978 and Dec. 1981. 160 babies (44%) needed breathing support (CNP, CPAP, IMV, IPPB). Pneumothorax occurred in 22 babies, 17 died. A severe cerebral

Hepatopulmonary syndrome and venous emboli causing intracerebral hemorrhages after liver transplantation: a case report.

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Increasing experience has fostered the acceptance of liver transplantation as a treatment for patients with hepatopulmonary syndrome. Morbidity and mortality is most commonly attributed to progressive arterial hypoxemia postoperatively. A cerebral hemorrhage has been reported in one patient with

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