fat necrosis/edema

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Intraarticular injection of arthritogenic factor causes mast cell degranulation, inflammation, fat necrosis, and synovial hyperplasia.

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Arthritis resembling human rheumatoid arthritis is produced in rats either by immunization with type II collagen or injection of complete Freund's adjuvant. The development of arthritis in both models may be mediated by a T cell-derived, type II collagen-specific protein that has been termed

Orbital fat necrosis following a revision endoscopic dacryocystorhinostomy

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Introduction: This report details a case of a preseptal fat necrosis in a 55-year old female, following a revision endoscopic dacryocystorhinostomy (DCR). Case description:

Transition of rat pancreatic juice edema into acute pancreatitis by single ethanol administration.

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The present study was done to determine the influence of single oral, intravenous and intraperitoneal ethanol administration at pre-existing pancreatic juice edema on frequency and severity of acute pancreatitis (AP). The rats were divided into six control (isolated treatment) and five study groups

Influence of pancreatic edema and short-term ischemia of rat pancreas on lipase- and alpha-amylase-activities in the serum and in the pancreas.

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Acute pancreatitis was produced in rats by a combination of dyschylic edema and short-term ischemia of the pancreas. The incidence of acute pancreatitis, reflected by peri- and extra-pancreatic fat necrosis, was found to be dependent on the duration of ischemia. Under most conditions there were no

[Subcutaneous inflammatory edema induced by MINE chemotherapy].

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BACKGROUND The MINE regimen (mitoguazone, ifosfamide, vinorelbine and etoposide) is a salvage chemotherapy for relapsed and refractory Hodgkin's disease. METHODS We report the cases of a 16-year-old girl and a 17-year-old boy who had Hodgkin's disease and developed painful and massive subcutaneous

Development of acute pancreatitis in rats after single ethanol administration and induction of a pancreatic juice edema.

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The effect of an acute gastral ethanol application and an additional induction of a pancreatic juice edema by an obstruction/hyperstimulation mechanism was studied in rats. Neither ethanol administration nor the combination of ductal obstruction and secretory stimulation resulted in an acute

Fat necrosis with features of erythema nodosum in a patient with metastatic pancreatic carcinoma.

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A 59-year-old man presented with painful subcutaneous nodules on the anterior surfaces of the legs. He had received oral antibiotics and supportive care for presumed cellulitis and thrombophlebitis, but had minimal improvement. Five months earlier, he had undergone pancreaticoduodenectomy for acinar

[Acute pancreatitis after treatment with sulindac].

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Two women, aged 91 and 46 years respectively, developed acute pancreatitis following treatment with sulindac (Clinoril) for five weeks in the one case and four and a half years in the other. The oldest patient became acutely ill. An emergency laparotomy was performed which showed considerable

Metaphyseal Core Decompression of the Distal Radius for Early Lunate Necrosis.

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Background: Metaphyseal core decompression of the distal radius (MCD) is clinically effective in early lunate necrosis without changing individual wrist mechanics. Its concept is based on the induction of physiologic mechanisms known as physiologic fracture healing response. However, this

Melatonin treatment of pigs with acute pancreatitis reduces inflammatory reaction of pancreatic tissue and enhances fitness score of pigs: experimental research.

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Severe acute pancreatitis is associated with high morbidity and mortality. Melatonin is known as the activator of antioxidant enzymes. The main purpose of this study was to evaluate the clinical effect of melatonin treatment in a pig model with induced acute

Bacterial peritonitis with hyperamylasemia in the absence of ultrastructural changes of pancreas. A light and electron microscopic study in the rat.

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Bacterial peritonitis was induced in rats by injecting two concentrations of E. coli (10(6) cfu/mL or 10(8) cfu/mL) and B. fragilis (10(8) cfu/mL or 5 x 10(8) cfu/mL) ip. Twenty-four hours later the serum amylase activity was significantly increased in both groups. At light microscopy scattered

Experimental acute pancreatitis induced by platelet activating factor in rabbits.

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This study indicates that a single injection of platelet activating factor (PAF, 50-500 ng) into the superior pancreaticoduodenal artery of rabbits induces dose-dependent morphologic alterations of pancreatic tissue and increases serum amylase levels, both consistent with the development of an acute

Pancreatic involvement in fatal human leptospirosis: clinical and histopathological features.

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Hyperamylasemia has been reported in more than 65% of patients with severe leptospirosis, and the true diagnosis of acute pancreatitis is complicated by the fact that renal failure can increase serum amylase levels. Based on these data we retrospectively analyzed the clinical and histopathological

The association of viral hepatitis and acute pancreatitis.

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The histological features of 24 pancreases obtained from patients who died of causes other than hepatitis, pancreatitis or pancreatic tumors, included a variable degree of autolysis, rare foci of inflammatory reaction but no hemorrhagic fat necrosis or destruction of elastic tissue in vessel walls

Effects of Tempol on Experimental Acute Necrotizing Pancreatitis Model in Rats.

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OBJECTIVE We aimed to investigate the effects of Tempol on local organ damage in an experimental acute pancreatitis model. METHODS This experimental study was conducted on 40 male Wistar- albino rats. The animals were randomly allocated into four groups: (i) Sham-operated group, laparotomies and

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