glutamate oxaloacetate transaminase/edema

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Neuroprotection by glutamate oxaloacetate transaminase in ischemic stroke: an experimental study.

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As ischemic stroke is associated with an excessive release of glutamate into the neuronal extracellular space, a decrease in blood glutamate levels could provide a mechanism to remove it from the brain tissue, by increasing the brain-blood gradient. In this regard, the ability of glutamate

Correlation between antiinflammatory and antiproteolytic properties of substituted benzylidenes.

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Twelve substituted benzylidenes were evaluated for antiinflammatory activity against carrageenin-induced edema in rats. The protection afforded by these compounds at a dose of 100 mg/kg, i.p., ranged from 30 to 60%. Sodium salicylate (100 mg/kg, i.p.), used as a reference drug, exhibited a 30%

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