goiter/hypoxia

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THE BIOASSAY OF THYROACTIVE MATERIALS BY THE MOUSE ANOXIA TEST AND GOITRE-PREVENTION RESPONSE IN THE SAME EXPERIMENTAL ANIMALS.

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[Changes of some indices of tissue metabolism in rabbits fed with strumigens for prolonged periods (concerning the role of hypoxia in pathogenesis of goiter].

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Genetic interaction analysis of VEGF-A rs3025039 and VEGFR-2 rs2071559 identifies a genetic profile at higher risk to develop nodular goiter.

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Nodular goiter in patients from areas of iodine deficiency is due to the growth of follicular and endothelial cells, involving different vascular-related growth factors in its pathogenesis.The aim of our study was to examine the association of known single

[Cytochrome oxidase activity of peripheral blood leukocytes in patients with diffuse toxic goiter].

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Patients with diffuse toxic goiter showed a distinct reduction of the cytochromoxidase activity in the peripheral blood leucocytes. The degree of reduction of the cytochromoxidase activity in the leucocytes depended on the severity of the clinical course and degree of increase of the thyroid gland.

Iodine-deficiency-induced long lasting angiogenic reaction in thyroid cancers occurs via a vascular endothelial growth factor-hypoxia inducible factor-1-dependent, but not a reactive oxygen species-dependent, pathway.

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BACKGROUND In the thyroid, iodine deficiency (ID) induces angiogenesis via a tightly controlled reactive oxygen species (ROS)-hypoxia inducible factor-1 (HIF-1)-vascular endothelial growth factor (VEGF) dependent pathway (ROS-HIF-VEGF). Deficient iodine intake may be associated with increased

Repeated episodes of respiratory distress in an obese parturient after cesarean delivery.

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A 25-yr-old obese parturient with mild asthma underwent an uneventful spinal anesthetic for primary cesarean delivery. Within 4 h after delivery, the patient twice developed acute shortness of breath, inspiratory stridor, and hypoxemia that required intubation. A battery of blood tests revealed no

An international delphi study of the causes of death and the criteria used to assign cause of death in bovine perinatal mortality.

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The objective of the present study was to elicit opinion from two groups of veterinarians [subject matter experts and non-subject matter experts] about the causes of bovine perinatal mortality and the criteria used to assign such causes. The subject matter experts were selected on the basis of their

18F-FDG uptake on PET in primary mediastinal non-thymic neoplasm: a clinicopathological study.

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BACKGROUND The usefulness of 2-[(18)F]-fluoro-2-deoxy-D-glucose ((18)F-FDG) positron emission tomography (PET) has been investigated in thymic epithelial tumors. However, little is known about PET imaging of (18)F-FDG in primary non-thymic mediastinal neoplasms. The aim of this study is to explore

The presence of adenosine A2a receptor in thyrocytes and its involvement in Graves' IgG-induced VEGF expression.

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Goitrogenesis in Graves' disease (GD) has been attributed to anti-TSH receptor antibody stimulation. Recently, a role for adenosine A2a receptor (A2aR) in goiter formation was reported in the thyroglobulin-A2aR transgenic mice. However, it is unclear whether A2aR is expressed in the thyroid and

Lipomatous lesions of the thyroid gland: a review.

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Described are rare thyroid lesions which either contain mature adipocytes or consist of lipid-rich follicle cells. Of the former, diffuse lipomatosis and adenolipoma of the thyroid are most likely to be derived from heterotopic islands of fat cells included in the gland during embryogenesis. On the

[Myocardial function and reactivity in thyrotoxicosis and in the post-thyrotoxic state (experimental-clinical study)].

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A response of the heart to hypoxia was studied in experimental thyrotoxicosis and physical exercise in patients with the thyrotoxic goiter. A decrease in the duration of cardiac hyperfunction and acceleration in the suppression of cardiac function in hypoxia were observed in rats with thyrotoxicosis

Recent insights into the cell biology of thyroid angiofollicular units.

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In thyrocytes, cell polarity is of crucial importance for proper thyroid function. Many intrinsic mechanisms of self-regulation control how the key players involved in thyroid hormone (TH) biosynthesis interact in apical microvilli, so that hazardous biochemical processes may occur without detriment

[Thyrotoxic crisis].

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A thyrotoxic Crisis is an acute or subacute metabolic explosion caused by uncontrolled release of tyronines from the thyroid gland (more frequently a diffuse thyroid gland) following its injury: strumectomy, administration of a therapeutic dose of I131, inflammations, or excessive stimulation

Expression of vascular endothelial growth factor and presence of angiovascular cells in tissues from different thyroid disorders.

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BACKGROUND Vascular endothelial growth factor (VEGF) is involved in tumor angiogenesis and other pathophysiological processes. METHODS We studied the localization of VEGF in human thyroid tissues to clarify its involvement in proliferative processes in a variety of thyroid disorders.

Expression of vascular endothelial growth factor (VEGF) in human thyroid tumors.

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Vascular endothelial growth factor (VEGF) is one of the factors related to angiogenesis of tumors and physiological and pathological proliferative processes. Location of VEGF in human thyroid tissue with various thyroid disorders was studied in order to explore its possible involvement in

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