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intracranial aneurysm/nicotine
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Roles of Nicotine in the Development of Intracranial Aneurysm Rupture.
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Background and Purpose- Tobacco cigarette smoking is considered to be a strong risk factor for intracranial aneurysmal rupture. Nicotine is a major biologically active constituent of tobacco products. Nicotine's interactions with vascular cell nicotinic acetylcholine receptors containing α7 subunits
The impact of hypertension and nicotine on the size of ruptured intracranial aneurysms.
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BACKGROUND
The goal of the study was to analyse retrospectively the impact of risk factors for subarachnoid haemorrhage (SAH) on the size of ruptured intracranial aneurysms in order to identify variables that might influence the discrepancy between average sizes of ruptured and unruptured
Women With First-Hand Tobacco Smoke Exposure Have a Higher Likelihood of Having an Unruptured Intracranial Aneurysm Than Nonsmokers: A Nested Case-Control Study
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Background: The prevalence of unruptured intracranial aneurysms (UIA) in females who smoke cigarettes and the association between smoking and hypertension with purely incidental UIAs have been unexplored.
Commentary: Women With First-Hand Tobacco Smoke Exposure Have a Higher Likelihood of Having an Unruptured Intracranial Aneurysm Than Nonsmokers: A Nested Case-Control Study
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Increased Incidence of Hypovitaminosis D Among Patients Requiring Treatment for Cerebral Aneurysms.
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OBJECTIVE
Aneurysmal subarachnoid hemorrhage management is hampered by our incomplete understanding of what variables promote aneurysm formation, growth, and rupture. Because hypovitaminosis D has been identified as a risk factor for other vascular diseases, we examined its association with cerebral
Risk of hemorrhage from de novo cerebral aneurysms.
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OBJECTIVE
A small percentage of patients will develop a completely new or de novo aneurysm after discovery of an initial aneurysm. The natural history of these lesions is unknown. The authors undertook this statistical evaluation a large cohort of patients with both ruptured and unruptured de novo
The clinical significance and reliability of self-reported smoking status in patients with intracranial aneurysms: a review.
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OBJECTIVE
Here we present a review of the pathophysiology of tobacco smoking on intracranial aneurysms, self-reported smoking status in these patients, screening tools and assays available for assessing active nicotine use, means of impacting smoking cessation rates, and the potential impact of
Smoking and non-smoking tobacco as risk factors in subarachnoid haemorrhage.
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OBJECTIVE
Swedish snuff is a particular form of non-smoking tobacco with high nicotine content. It is unknown whether this form of tobacco is a risk factor similar to smoking for suffering subarachnoid haemorrhage (SAH). In the present study we report our finding concerning smoking and snuff as risk
Effects of tobacco dose and length of exposure on delayed neurological deterioration and overall clinical outcome after aneurysmal subarachnoid hemorrhage.
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OBJECTIVE
The association between smoking and intracranial aneurysms is now well recognized. However, the relationship between tobacco use and outcome after aneurysmal subarachnoid hemorrhage (SAH) is not as well understood and published results are contradictory. The purpose of this study is to
Epidemiology of intracranial aneurysms of Mississippi: a 10-year (1997-2007) retrospective study.
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BACKGROUND
Despite massive efforts, progress so far has been modest in isolating the genetic determinants for intracranial aneurysm (IA). More detailed epidemiology data might be essential for successful genome-wide association study. Here, we aimed to investigate epidemiology and identify the key
Influence of smoking, hypertension, and sex on the phenotypic expression of familial intracranial aneurysms in siblings.
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OBJECTIVE
To investigate the effects of smoking, hypertension, and sex on the phenotypic expression of familial intracranial aneurysms (FIAs).
METHODS
We retrospectively reviewed the case records of 806 consecutive patients undergoing aneurysm surgery at our institution (1986-1995) and discovered 24
Smoking Does Not Affect Occlusion Rates and Morbidity-Mortality after Pipeline Embolization for Intracranial Aneurysms.
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Smoking is a major risk factor for patients with intracranial aneurysms, yet the effects of smoking on outcomes of aneurysm with flow-diverter treatment remain unknown. We studied the impact of smoking on long-term angiographic and clinical outcomes after flow-diverter treatment of intracranial
A new comorbidities index for risk stratification for treatment of unruptured cerebral aneurysms.
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OBJECTIVE Comorbidities have an impact on risk stratification for outcomes in analyses of large patient databases. Although the Charlson Comorbidity Index (CCI) and the Elixhauser Comorbidity Index (ECI) are the most commonly used comorbidity indexes, these have not been validated for patients with
Cerebral Aneurysms Differ in Patients with Hysterectomies.
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OBJECTIVE
Female sex is a well-known risk factor for aneurysm formation. Although the role of the ovaries and estrogen in aneurysm development has been supported, other organ-hormone pairs unique between sexes also may be implicated. In this study, we aimed to determine whether intracranial
Smokers with intracranial aneurysms who underwent neurosurgical or endovascular treatment have not stopped smoking one year after surgery.
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Smoking increases the risk of forming, growing, and rupture of intracranial aneurysms. We retrospectively reviewed patients with intracranial aneurysms treated by neurosurgical or endovascular treatment--154 patients (45 men, 109 women, 15 to 62 years, average 46.3 years, CI +/- 1.72). We found 74%
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