mastocytosis/albumin

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Repeated antigen challenge in rats induces a mucosal mast cell hyperplasia.

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BACKGROUND A link between mast cells and a number of intestinal diseases has been suggested. Treatment of adult rats with connective tissue mast cell degranulating agents has been shown to induce an intestinal mucosal mast cell hyperplasia. We have examined the hypothesis that repeated systemic

Next-generation sequencing in systemic mastocytosis: Derivation of a mutation-augmented clinical prognostic model for survival.

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In routine practice, the World Health Organization classification of systemic mastocytosis (SM) is also the de facto prognostic system; a core value is distinguishing indolent (ISM) from advanced SM (includes aggressive SM [ASM], SM with associated hematological neoplasm [SM-AHN] and mast cell

Markers/parameters for the evaluation of natural resistance status of small ruminants against gastrointestinal nematodes.

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The high prevalence of anthelmintic-resistant gastrointestinal nematodes (GINs) throughout the world has led to the need for alternative worm control strategies. One of the possible substitutes to reduce the problems of drug resistance and residue is the evaluation/breeding of small ruminants for

Heparin increases the adhesion of murine mammary adenocarcinoma cells (LM3). Correlation with the presence of heparin receptors on cell surface.

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Mastocytosis is a common feature around solid tumors. Due to mast cell (MC) degranulation, heparin and other chemical mediators are released to surrounding tissues. The aim of this paper is to investigate the role of heparin and chemically modified heparins, on a murine mammary adenocarcinoma cell

Mast cells in a murine lung ischemia-reperfusion model of primary graft dysfunction.

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Primary graft dysfunction (PGD), as characterized by pulmonary infiltrates and high oxygen requirements shortly after reperfusion, is the major cause of early morbidity and mortality after lung transplantation. Donor, recipient and allograft-handling factors are thought to contribute, although new

Aurora kinase inhibitor tozasertib suppresses mast cell activation in vitro and in vivo.

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Mast cells (MCs) are important in allergic reactions. Here, we assess the anti-allergic effects of the anti-cancer drug tozasertib specifically regarding regulatory effects on MCs activation.Tozasertib effects on MC degranulation was determined by measuring

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