megacolon/carbohydrate

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Nutrition and ulcerative colitis.

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The role of diet in the aetiology and pathogenesis of ulcerative colitis (UC) remains uncertain. Impaired utilization by colonocytes of butyrate, a product of bacterial fermentation of dietary carbohydrates escaping digestion, may be important. Sulphur-fermenting bacteria may be involved in this

The influence of diet and dimethylhydrazine on the small and large intestine of vervet monkeys.

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The morphological features of the intestine in monkeys on various diets with and without carcinogen were studied. Seventy adult female vervet monkeys were divided into seven treatment groups. Four groups received a Western high-fat low fibre diet (WD); two a Prudent low-fat higher fibre diet (PD)

Chagasic enteropathy.

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Involvement of the hollow organs of the digestive apparatus can occur in patients in the chronic phase of Chagas' disease. The basic mechanism is destruction of neurons of the enteric nervous system. Whereas megaesophagus and megacolon are the most notable and most extensively studied expressions of

[Development of Wernicke's encephalopathy during the period of oral food intake after a subtotal colectomy for ulcerative colitis].

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A 61-year-old woman was hospitalized because of ulcerative colitis which had caused fever, vomiting and diarrhea since June 16, 1992. Then she developed toxic megacolon, and was transferred to our hospital on the 1st of July and underwent subtotal colectomy the same day. After surgery, she received

ABO, Secretor and Lewis histo-blood group systems influence the digestive form of Chagas disease.

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Chagas disease, caused by Trypanosoma cruzi, can affect the heart, esophagus and colon. The reasons that some patients develop different clinical forms or remain asymptomatic are unclear. It is believed that tissue immunogenetic markers influence the tropism of T. cruzi for different organs. ABO,

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