myocardial stunning/necrosis

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Contractile reserve and extent of transmural necrosis in the setting of myocardial stunning: comparison at cardiac MR imaging.

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OBJECTIVE To perform a comparison of cardiac magnetic resonance (MR) imaging-derived ejection fraction (EF) during low-dose dobutamine infusion (EF(D)) with the extent of segments with transmural necrosis in more than 50% of their wall thickness (ETN) for the prediction of major adverse cardiac

Non-invasive cardiovascular imaging for myocardial necrosis, viability, stunning and hibernation: evidence from an umbrella review encompassing 12 systematic reviews, 286 studies, and 201,680 patients

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Introduction: The concomitant presence of myocardial necrosis with myocardial ischemia, stunning and hibernation may complicate appraisal of left ventricular (LV) function and patient management. Several imaging modalities have been

[Pathophysiological evolution of coronary (ischemic) heart disease: ischemia, dystrophy, necrosis, sclerosis].

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Pathophysiological evolution of coronary (ischemic) heart disease in the following order: ischemia--dystrophy--necrosis (infarction)--myocardial sclerosis is postulated. The authors give clinical, instrumental, laboratory characteristics of ischemia, focal dystrophy with 2 types of manifestations:

Do neutrophils contribute to myocardial stunning?

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Although removal of neutrophils from the arterial blood by mechanical filtration has been reported to prevent or reduce the severity of myocardial stunning caused by a 15 minute coronary artery occlusion in the dog, neutrophil filtration does not protect against myocardial dysfunction following a

Myocardial stunning following no flow ischaemia is diminished by levosimendan or cariporide, without benefits of combined administration.

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OBJECTIVE Levosimendan, a calcium sensitiser, and cariporide, a blocker of the Na+/H+ exchanger, decrease necrosis and improve function following myocardial ischaemia. However, their role in myocardial stunning is unclear. We tested the hypothesis that levosimendan, cariporide, or their combination

Increased myocardial tumor necrosis factor-alpha in a crystalloid-perfused model of cardiac ischemia-reperfusion injury.

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BACKGROUND The heart is a tumor necrosis factor-alpha (TNF-alpha)-producing organ. Recent basic experimental and clinical evidence suggests that TNF-alpha is an important mediator of myocardial injury during acute myocardial infarction, chronic heart failure, cardiac allograft rejection, and

[Coronary artery spasm as a cause of perioperative myocardial infarction and stunned myocardium].

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We report a male patient who had recurrent coronary artery spasm on withdrawal from cardiopulmonary bypass, which led to myocardial infarction and stunned myocardium. The spasm responded to conventional medication. Transesophageal echocardiogram showed no remarkable asynergy during the operation.

[The stunned myocardium: ischemia-induced reversible myocardial dysfunction].

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The "stunned" myocardium is the result of an ischemic insult which is not of sufficient severity to produce myocardial necrosis but affects myocardial function, biochemical processes and ultrastructure for a prolonged period of time. A "stunned" myocardium can be expected when preoperative asynergic

Thallium 201 kinetics in stunned myocardium characterized by severe postischemic systolic dysfunction.

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The hypothesis tested in this study was that despite the presence of severe postischemic myocardial dysfunction ("stunning"), the extraction and subsequent intracellular washout of thallium 201 should be preserved as long as irreversible sarcolemmal membrane injury was avoided. To produce myocardial

Preconditioning does not attenuate myocardial stunning.

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BACKGROUND Despite numerous reports that one or more episodes of brief coronary artery occlusion preconditions the myocardium and dramatically reduces myocardial infarct size produced by a subsequent prolonged ischemia, we recently demonstrated that preconditioning does not attenuate contractile

Myocardial stunning in dogs: preconditioning effect and influence of coronary collateral flow.

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In open-chest dogs the left anterior descending (LAD) coronary artery diagonal branch was encircled with a pneumatic occluder. Pairs of ultrasonic crystals were inserted into LAD myocardium and remote normal muscle. The coronary artery was occluded for 5 minutes, followed by 10 minutes of

Can myocardial stunning contribute to myocardial infarction during coronary artery bypass surgery?

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Myocardial stunning commonly occurs after cardiopulmonary bypass (CPB). Myocardial stunning can be cardioprotective under some conditions, but in other situations may actually contribute to myocardial infarction (MI). Vascular endothelial stunning may be one of the mechanisms by which myocardial

The role of nuclear cardiac imaging in redefining neurogenic stunned myocardium in subarachnoid hemorrhage: a deeper look into the heart.

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Subarachnoid hemorrhage may be complicated by neurogenic stunned myocardium, a catecholamine-induced transient cardiomyopathy that displays a wide clinical spectrum of cardiac abnormalities, including electrocardiographic changes, arrhythmias, myocardial necrosis, and left ventricular systolic and

[Coronary perfusion, necrosis area, pumping function of the myocardium, and cardiac electrical stability following revascularisation of infarction-affected myocardium].

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Analysing the literature data suggests that coronary angioplasty in acute myocardial infarction (AMI) fails to completely remove the focus of ischaemia, even in case of successful recanalization of the infarction-related artery. Inadequate post-angioplasty reperfusion of the infarction-stunned

The phenomenon of "stunned" myocardium: implications for coronary care nurses.

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In recent years, the use of thrombolytic therapy for the treatment of acute myocardial infarction has become the standard of care. A new phenomenon known as "stunned" myocardium has been described with the use of this treatment. Stunned myocardium refers to tissue that has been reperfused before of

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