oligohydramnios/potasyòm

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Renal function in angiotensinogen gene-mutated renal tubular dysgenesis with glomerular cysts.

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BACKGROUND Inherited renal tubular dysgenesis (RTD) is caused by mutations in the genes encoding the components of the renin-angiotensin system (RAS). RTD is characterized by oligohydramnios, renal failure, neonatal hypocalvaria, and severe hypotension. The histological characteristics, underlying

A 3 year, prospectively-designed study of late selective multifetal pregnancy reduction.

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The aim of our study was to evaluate the pregnancy outcomes of late selective multifetal reduction (MFPR). We performed a 3 year, prospectively-designed study in which 28 patients underwent MFPR at a mean gestational age of 20.2 +/- 3.9 weeks (range 14-29 weeks). The indications for MFPR included:

Renal dysgenesis in a monozygotic twin: association with in utero exposure to indomethacin.

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We report oligohydramnios and renal dysgenesis in one of identical twins, which might have resulted from in utero exposure to early, prolonged high-dose indomethacin. The proposita was the second of twin girls born at 36 weeks of gestation. Pregnancy was complicated initially by polyhydramnios in

Early fetal obstructive uropathy produces Potter's syndrome in the lamb.

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BACKGROUND If creating an obstructive uropathy early in glomerulogenesis produces MCDK (Multicystic Dysplastic Kidney), then a very early obstruction may produce Potter's Syndrome (PS) with oligohydramnios. METHODS Fetal lambs at 50 days' gestation underwent urethral and urachal ligation using fine

Renal tubular acidosis in pregnancy: case report and literature review.

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Renal tubular acidosis is a rare form of chronic metabolic acidosis, which is either inherited as an autosomal dominant condition (Types 1, 2, and 3) or acquired. Its effects on pregnancy and vice versa are not known, but chronic acidosis may affect fetal bone growth and development. Chronic

Outcome of Two Pregnancies in a Patient with Gitelman's Syndrome-A Case Report.

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> Gitelman's syndrome is primary renal tubular hypokalemic metabolic alkalosis with hypocalciuria and magnesium deficiency. We present the prenatal course and outcome of two pregnancies in a patient with Gitelman's syndrome. The complication encountered was oligohydramnios. Close monitoring of serum

Hyperechogenic kidneys and polyhydramnios associated with HNF1B gene mutation.

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HNF1B mutation is the leading cause of isolated hyperechogenic fetal kidneys with normal or moderately large size. Although most cases have normal amniotic fluid volume, some cases present with early oligohydramnios and renal failure associated with high perinatal mortality. Here we report on seven

Effects of inhibition of the maternal renin-angiotensin system on maternal and fetal responses to drainage of fetal fluids.

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To find out which of the effects of angiotensin converting enzyme (ACE) inhibitors on the fetus are due to their actions in the mother and which are direct effects due to blockade of the fetal renin-angiotensin system, enalapril (150 mg twice daily i.v.), which does not readily cross the sheep

Maternal dehydration: impact on ovine amniotic fluid volume and composition.

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Maternal dehydration consistent with mild water deprivation or moderate exercise results in maternal and fetal plasma hyperosmolality and increased plasma arginine vasopressin (AVP). Previous studies have demonstrated a reduction in fetal urine and lung fluid production in response to maternal

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