poliomyelitis/phosphatase

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Comparative biochemical studies on normal and on poliomyelitis virus-infected tissue cultures. V. Profund alteration of acid and alkaline phosphatase activity in infected rhesus kidney cells.

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Experimental evidence is presented for drastic changes in phosphomonoesterase activities of tissue cultures, brought about by infection with poliomyelitis viruses. Acid phosphatase activity went through a maximum before decreasing almost to zero level. Alkaline phosphatase activity diminished

[ACID AND ALKALINE PHOSPHATASE IN SPINAL CORD NEURONS OF MONKEYS IN NORMAL CONDITIOSN AND IN EXPERIMENTAL POLIOMYELITIS].

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Serum alkaline phosphatase in monkeys and man during poliomyelitis.

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Stable isotope labeling by amino acids in cell culture and differential plasma membrane proteome quantitation identify new substrates for the MARCH9 transmembrane E3 ligase.

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The regulation of cell surface receptor expression is essential for immune cell differentiation and function. At the plasma membrane ubiquitination is an important post-translational mechanism for regulating expression of a wide range of surface proteins. MARCH9, a member of the RING-CH family of

Rimmed vacuoles with beta-amyloid and ubiquitinated filamentous deposits in the muscles of patients with long-standing denervation (postpoliomyelitis muscular atrophy): similarities with inclusion body myositis.

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In the chronically denervated muscles of patients with prior paralytic poliomyelitis, there are secondary myopathic features, including endomysial inflammation and rare vacuolated fibers. To assess the frequency and characteristics of the vacuoles and their similarities with those seen in inclusion

Enterovirus infection may induce humoral immune response reacting with islet cell autoantigens in humans.

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Molecular mimicry is one of the mechanisms by which enterovirus infections have been postulated to have a role in the pathogenesis of type 1 diabetes. Immunogenic epitopes in enterovirus capsid protein VP1 and procapsid protein VP0 have sequence similarities with diabetes-associated epitopes in

Poliovirus entry into human brain microvascular cells requires receptor-induced activation of SHP-2.

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Viruses use specific receptor molecules to bind selectively to target cells. Receptors have often been considered as mere docking sites, but they may also possess intrinsic signaling capacities that serve to prime the cell for entry and infection. Poliovirus (PV) initiates infection by binding to

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