pulmonary atresia/lafyèv

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Presumptive Q fever myocarditis associated with Coxiella burnetii infection of a homograft valve in the outflow tract of the right ventricle: review and case report.

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A man with pulmonary atresia and a subaortic ventricular septal defect had a radical surgical repair at the age of 16 years with correction of pulmonary atresia by replacement with an aortic valve homograft. This later became infected with Coxiella burnetii. He died at the age of 27 years from renal

A case of bacteremia and disseminated intravascular coagulation after the conduit procedure for tetralogy of Fallot with pulmonary atresia.

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A case undergoing conduit procedure for tetralogy of Fallot with pulmonary atresia was complicated postoperatively by bacteremia due to non-fermentative Gram-negative rods and by disseminated intravascular coagulation. He was able to be cured without any sequela. The patient was a 16-year-old male,

Ductus arteriosus dilatation by prostaglandin E1 in infants with pulmonary atresia.

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Infants with pulmonary atresia depend on patency of the ductus arteriosus for survival in the immediate postnatal period. Despite continuing hypoxemia after birth the ductus arteriosus usually constricts, thus reducing pulmonary blood flow. This often occurs while awaiting surgical palliation or

Prolonged prostaglandin E1 therapy in a neonate with pulmonary atresia and ventricular septal defect and the development of antral foveolar hyperplasia and hypertrophic pyloric stenosis.

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Prostaglandin E1 (alprostadil) is widely used for maintaining the patency of ductus arteriosus in ductus-dependent congenital heart defects in neonates to improve oxygenation. Among more common side effects are fever, rash, apnoea, diarrhoea, jitteriness, and flushing. More severe side effects are

Necrotizing arteritis in uncorrected tetralogy of Fallot with pulmonary artery.

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A 10-year-old girl with uncorrected tetralogy of Fallot with pulmonary atresia presented with fevers of unknown origin and left lung infiltrates. At autopsy, necrotizing vascular changes resembling those of severe pulmonary hypertension (grade VI in the Health-Edwards classification) were confined

[Brain abscess in congenital cyanotic heart defect in adulthood].

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A 27-year-old patient with pulmonary atresia, ventricular septal defect, and multifocal lung-perfusion suddenly developed headache and left facial sensory disturbances. Using computed tomography and magnetic resonance imaging a temporo-parietal brain abscess was diagnosed. After abscess aspiration

Prostaglandin E1: first stage palliation in neonates with congenital cardiac defects.

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E-type prostaglandins (PGE1) can effectively maintain the patency of the ductus arteriosus in neonates. Its use, therefore can be life saving in infants born with ductus dependent congenital heart disease. Although PGE1 is available for over two decades in western world, it has been introduced in

Reappraisal of the prostaglandin E1 dose for early newborns with patent ductus arteriosus-dependent pulmonary circulation.

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OBJECTIVE The usual initial dose of prostaglandin E1 (PGE1) for ductal-dependent congenital heart disease (CHD) is 50-100 ng/kg/minute. The aim of this study was to review our experience of a low initial dose of PGE1 treatment in early newborns with congenital heart disease and patent ductus

Infected pseudoaneurysm following a modified Blalock-Taussig shunt procedure.

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A 3-year-old boy with pulmonary atresia with ventricular septal defect, who had undergone placement of a modified Blalock-Taussig shunt, presented with a 1-week history of high fever. Computed tomography showed a pseudoaneurysm at the anastomosis between the right brachiocephalic artery and the

[Bacterial endocarditis in childhood].

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BACKGROUND Bacterial endocarditis in childhood is a rare but serious disease. The group of children with congenital heart disease at risk to develop bacterial endocarditis increases, because more children survive with advanced medical and surgical management. Rheumatic Fever as predisposing heart

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