purpura fulminans/seizures

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[A 52-year-old woman with acute shock and purpura fulminans. Pneumococcal sepsis].

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We report a 52-year-old female patient admitted with fever, chills, and myalgias since the previous day. On the day of admission she had a generalized seizure. The patient had no previous illnesses. Laboratory investigations showed consumptive coagulopathy with clinical manifestations of shock and

Dilantin-induced disseminated intravascular coagulation with purpura fulminans. A case report.

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A patient developed disseminated intravascular coagulation with purpura fulminans 1 month after starting Dilantin therapy for a seizure disorder. In addition, the patient developed exfoliative dermatitis, hepatitis, cutaneous vasculitis, and microangiopathic hemolytic anemia. She was successfully

Nursing care of a child with meningococcemia.

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This case presentation will discuss the pathophysiology of a child in septic shock due to Neisseria meningitidis. The most prevalent nursing care concerns of this case encountered during the pediatric intensive care unit (PICU) and during the general floor stay will be addressed. The nursing skill

Meningococcal infections in children: a review of 100 cases.

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One hundred children with meningococcal infection diagnosed from January 1, 1985, to February 29, 1988, were reviewed. Clinical manifestations ranged from fever alone to fulminant septic shock with purpura fulminans. Twenty-nine percent of the children presented without skin lesions. Of the 55

Fetal hydrocephalus and neonatal stroke as the first presentation of protein C deficiency.

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Severe protein C-deficiency is a rare heritable thrombophilia of the newborn. Infants with biallelic PROC mutations present purpura fulminans and intracranial thromboembolism, while the prenatal onset of mutated heterozygotes remains unclear. We herewith present the first case of fetal

A case of protein C deficiency associated with cerebral infarction and obstruction of deep leg and inferior mesenteric veins.

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Protein C, a vitamin K-dependent protein, is a blood coagulation inhibitor. Its deficiency causes systemic thrombosis. A 31-year-old woman developed cerebral infarction followed by late psychomotor seizures, and thrombosis in the inferior mesenteric vein and bilateral crural veins. Her parents were

Cerebral palsy in siblings caused by compound heterozygous mutations in the gene encoding protein C.

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We report two sisters with extensive bilateral periventricular haemorrhagic infarction (PVHI) causing cerebral palsy (CP). The older sister presented at 20 months with cortical visual blindness, spastic diplegia, and purpura fulminans. The younger sister presented aged 3 days old with apnoeas and

Fulminant meningococcemia and acute renal failure in a 3-year-old boy.

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Acute renal failure is a common occurrence in sepsis, but is rarely reported in meningococcemia. We present a young child diagnosed with fulminant meningococcemia who had several poor prognostic factors, including hypotension, thrombocytopenia, purpura fulminans, seizures, the absence of meningitis

Invasive meningococcal infection: analysis of 110 cases from a tertiary care centre in North East India.

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OBJECTIVE To report an outbreak of invasive meningococcal disease from Meghalaya, in the north east India, from January 2008 through June 2009. METHODS Retrospective review of case sheets was done. One hundred ten patients with invasive meningococcal disease were included for the study. RESULTS Of

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