retinopathy of prematurity/hypoxia

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The Impact of Hypoxemia on the Development of Retinopathy of Prematurity in Infants Less Than 29 Weeks of Gestation.

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OBJECTIVE To study the impact of cumulative exposure to hypoxemia on the development of retinopathy of prematurity (ROP) in preterm infants less than 29 weeks' gestation. METHODS This is a retrospective analysis of the effect of cumulative exposure to hypoxemia during the first 10 weeks of life in

Hypoxia-induced retinal neovascularization in zebrafish embryos: a potential model of retinopathy of prematurity.

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Retinopathy of prematurity, formerly known as a retrolental fibroplasia, is a leading cause of infantile blindness worldwide. Retinopathy of prematurity is caused by the failure of central retinal vessels to reach the retinal periphery, creating a nonperfused peripheral retina, resulting in retinal

Role of hypoxia during normal retinal vessel development and in experimental retinopathy of prematurity.

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OBJECTIVE To test the hypothesis that retinal hypoxia is present during vascular development in normal rat pups and in a newborn rat model of retinopathy of prematurity (ROP). METHODS Preretinal vitreous PO(2) measurements were made during room air breathing using (19)F magnetic resonance

The relationship between patterns of intermittent hypoxia and retinopathy of prematurity in preterm infants.

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BACKGROUND We have previously shown an increased incidence of intermittent hypoxemia (IH) events in preterm infants with severe retinopathy of prematurity (ROP). Animal models suggest that patterns of IH events may play a role in ROP severity as well. We hypothesize that specific IH event patterns

Beta-adrenoreceptor agonism influences retinal responses to hypoxia in a model of retinopathy of prematurity.

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OBJECTIVE In a mouse model of oxygen-induced retinopathy (OIR), a well-established model of retinopathy of prematurity (ROP), blocking beta-adrenoreceptors (β-ARs), and, in particular, β2-ARs, counteracts retinal responses to hypoxia. In the present work, we determined the effects of the β-AR

[Ocular malformations in offspring of rats subjected to anoxemia during pregnancy with special reference to the problem of retrolental fibroplasia in man].

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Experimental approach to the pathogenesis of retrolental fibroplasia. III. Changes in the eye induced by exposure of newborn mice to general hypoxia.

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Hypoxemia and retinopathy of prematurity.

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Retrolental fibroplasia and anoxia.

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ANOXIA and retrolental fibroplasia.

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Retrolental fibroplasia--associated with intrauterine anoxia?

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Congenital malformations of the eye induced in mice by maternal anoxia; with particular reference to the problem of retrolental fibroplasia in man.

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[Effect and correlation of various risk factors in the development of retinopathy of prematurity. A retrospective study of 338 premature infants].

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The statistical significance of ophthalmological and pediatric findings in the development of retinopathy of prematurity (ROP) was evaluated in a retrospective study of 338 premature infants. Short gestation time, frequent hypoxia, and low birthweight were strongly correlated with advanced stages of

Hot Topics in Retinopathy of Prematurity.

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Retinopathy of prematurity (ROP) is a condition seen in premature infants that is characterized by abnormal retinal blood vessel growth incited by relative hyperoxia and followed by hypoxia. It can have severe consequences ranging from high myopia to blindness. This article reviews recent "hot"

Safety of Retinopathy of Prematurity Examination and Imaging in Premature Infants.

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OBJECTIVE To describe adverse events (AEs) and noteworthy clinical or ocular findings associated with retinopathy of prematurity (ROP) evaluation procedures. METHODS Descriptive analysis of predefined AEs and noteworthy findings reported in a prospective observational cohort study of infants <1251 g

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