rheumatic fever/phosphatase

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[Changes in blood calcium, phosphorus and alkaline phosphatase levels in rheumatoid polyarthritis and other types of inflammatory rheumatism].

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Albuminemia, calcemia, phosphoremia and alcaline phosphatasemia were measured in three groups of 52 subjects each : rheumatoid arthrits, inflammatory rheumatisms other than rheumatoid arthritis and lumbarthrosics serving as a reference group. Calcemia and albuminemia were significantly lower in

[Genetic variants of acid erythrocytic phosphatase as a risk factor of rheumatic fever].

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A study was made of the genetic variants (isoenzymes, phenotypes) of acid erythrocytic phosphatase (AcP) in 120 patients with rheumatic fever. There were 78 women and 42 men aged 16 to 57 years. The population data concerned with distribution of the AcP variants among the population of Moscow were

[Alkaline phosphatase activity in granular polynuclear neutrophils in children with rheumatic fever].

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[Beta-glucuronidase and acid phosphatase activity in children with rheumatic fever].

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Children with and without rheumatic fever. III. Blood serum vitamins and phosphatase data.

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[BEHAVIOR OF ALKALINE PHOSPHATASE IN PERIPHERAL BLOOD LEUKOCYTES IN CHILDREN WITH RHEUMATIC FEVER].

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Alkaline phosphatase of polynuclear neutrophils in children with rheumatic fever.

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Hepatotoxicity of high dose salicylate therapy in acute rheumatic fever.

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Liver function tests, including serum alanine aminotransferase (ALT) activity, serum bilirubin, alkaline phosphatase, serum proteins, blood ammonia levels and intravenous glucose utilization, were monitored in 50 children with acute rheumatic fever receiving anti-rheumatic doses of aspirin. There

Distribution of cells bearing B-cell alloantigen(s) in North Indian rheumatic fever/rheumatic heart disease patients.

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Numerous investigators have developed monoclonal antibodies against B-cell alloantigen(s) of rheumatic fever. However, the developed monoclonals do not have the same significance in all the populations. We have developed a battery of monoclonals against B-cell alloantigens of North Indian rheumatic

Serum cholesterol levels in patients with acute rheumatic fever.

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OBJECTIVE To determine abnormalities of cholesterol metabolism in children with acute rheumatic fever (ARF). METHODS Cross-sectional study and prospective study. METHODS Pediatric ward of the Khon Kaen (Thailand) University Hospital. METHODS Forty-six confirmed patients with ARF (during the acute

Gliotoxin potentiates osteoblast differentiation by inhibiting nuclear factor-κB signaling.

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The differentiation of pluripotent mesenchymal stem cells to mature osteoblasts is crucial for the maintenance of the adult skeleton. In rheumatic arthritis, osteoblast differentiation is impaired by the overproduction of cytokine tumor necrosis factor (TNF)‑α. It has been demonstrated that TNF-α is

No association of PTPN22 R620W gene polymorphism with rheumatic heart disease and systemic lupus erythematosus.

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Rheumatic heart disease (RHD) or acute rheumatic fever (ARF) develops as a consequence of an exaggerated immune response to Group A beta haemolytic streptococci causing pharyngitis. The molecular mimicry appears between human cardiac myosin and M protein of group A streptococcal membranes. The

[Cyclic nucleotides and enzymes of the synovial fluid in various rheumatic diseases].

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The authors measured the activities of four enzymes (L.D.H., acid phosphatase, B glucuronidase, and lysozyme) and the contents of AMPc and GMPc in the synovial fluid in 35 patients with rheumatic disease. In those with various forms of inflammatory rheumatism, they found negative correlation between

[Zinc and enzymes in the synovial fluid and blood in various types of rheumatism].

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It appears that in rheumatoid arthritis and, to a lesser extent, in the other forms of inflammatory rheumatism, the level of zinc in the blood serum is lowered, whereas synovial zinc is increased. In the synovial fluid, there is a very significant correlation between enzyme activity and the

[New research on synovial enzymology].

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After reviewing previous work on the subjects, the authors show that the synovial fluid in subjects with inflammatory rheumatism contained higher levels of lysozyme and of beta-glucuronidase comparable with those of the acid phosphatases and of lacto-dehydrogenase that they were interested in

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