serum sickness/albumin

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Serum sickness nephropathy in the rat using cationized albumin: effect of preimmunization and antigen dose.

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A model of glomerulonephritis induced in preimmunized rats with cationic albumin is described. Extensive glomerular immune complex formation and a severe nephrotic syndrome occurred within 5 days of commencement of daily intravenous injections. Severity of disease was markedly influenced by the

Serum sickness due to bovine serum albumin sensitization during in vitro fertilization.

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Of 32 women included in the in vitro fertilization (IVF) program in our hospital in 1987, in whom a medium containing bovine serum albumin (BSA) (Menezo's medium) was employed for rinsing follicles, 5 (15%) developed a symptom complex compatible with serum sickness within 8-12 days after oocyte

The effects of chronic serum sickness on albumin distribution and glucose utilization in rat brain.

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The level of cerebrospinal fluid (CSF) protein is elevated in diseases and disease models that are associated with circulating immune complexes such as serum sickness. Circulatory immune complexes are known to deposit in the basal lamina of fenestrated capillaries and may, as a result, affect both

Bovine serum albumin chronic serum sickness nephropathy in rats.

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We administered bovine serum albumin (BSA) 1 or 3 mg i.v. into hyper-immune Sprague-Dawley rats weekly for up to 6 months. Animals with free circulating antibody 1 h after BSA developed mesangial deposits of IgG and C-3 without proteinuria. Rats without free antibody at 1 h developed either

Antinephritic effect of prostaglandin E1 on serum sickness nephritis in rats (5). Effect of PGE1 on disposal of heat-aggregated bovine serum albumin in the glomerulus.

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In the present study, we investigated whether prostaglandin E1 (PGE1) could accelerate the disposal of heat-aggregated BSA (a-BSA) in the glomerulus by mesangial cells and/or resident mesangial cells. ICR mice were injected i.v. with 90 mg/100 g B.W. of a-BSA 3 times at 4-hr intervals. Kidneys were

Immunophysiology of serum sickness; alterations in the blood volume and thiocyanate space in relation to humoral antibodies in rabbits sensitized with human albumin or globulin.

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Role of aggregated bovine albumin in pathogenesis of serum sickness nephritis in rabbits.

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Serum sickness due to crystallized bovine albumin.

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[Serum sickness linked to in vitro fertilization protocols. The responsibility of bovine serum albumin].

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Validation of the PEG-IgG screening test for soluble immune complexes by longitudinal studies in experimental acute serum sickness.

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A wide variety of tests for the detection of circulating immune complexes (IC) has been proposed by different authors, but there is very little to no information concerning the performance of IC screening assays in samples known to contain in vivo-formed IC. The purpose of our investigation was to

Intra-ocular and CNS listeriosis following systemic infection in Wistar rats with chronic serum sickness.

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The relationship of immune complex deposits and cell-mediated immunity to the occurrence of ocular and brain listeriosis following a low (0.007-0.03 LD50) intraperitoneal challenge was investigated in 68 Wistar rats with chronic experimental serum sickness and 68 normally resistant controls. The

The role of polymorphonuclear leukocytes in rat chronic serum sickness-type nephritis.

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In order to investigate the role of polymorphonuclear leukocytes (PMN) in the distribution of antigen in various organs and in the development of nephritis, chronic serum sickness-type nephritis was induced in both anti-rat PMN rabbit serum (APS)-treated and normal rabbit serum (NRS)-treated rats by

Deposition of circulating antigen--antibody complexes in the gastrointestinal tract of rabbits with chronic serum sickness.

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The possible role of circulating immune complexes (IC) in the production gastrointestinal lesions was studied in rabbits with chronic serum sickness (CSS) induced by multiple daily injections of bovine serum albumin (BSA). All rabbits generating a marked antibody response developed IC

Chronic serum sickness glomerulonephritis: removal of glomerular antigen and electron-dense deposits is largely dependent on plasma complement.

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Chronic serum sickness glomerulonephritis was induced in 20 rats, using radio-labelled cationised bovine serum albumin (BSA) as antigen. Four days after the last dose of antigen, half the rats were given cobra venom factor (CVF) in doses sufficient to render plasma complement activity undetectable.

Effect of removal of cholesterol diet upon serum sickness-cholesterol-induced atherosclerosis.

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In this report, the role of vascular allergy (i.e., hypersensitivity) in the potentiation of atherogenesis has been studied. In order to accomplish this, bovine serum albumin (BSA) was administered to rabbits in quantities sufficient to cause the occurrence of serum sickness (a type of

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