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Rotavirus-like, calicivirus-like, and 23-nm virus-like particles associated with diarrhea in young pigs.

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Virus particles morphologically similar to caliciviruses and rotaviruses were detected by electron microscopy (EM) in the intestinal contents of a 27-day-old diarrheic nursing pig. A third small spherical 23-nm virus-like particle was also observed. Calicivirus-like particles averaged 33 nm in

Serological response in cholera-like diarrheas.

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Diarrhea caused by a Cyanobacterium-like organism.

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Seven cases of watery diarrhea of explosive onset, three of them ending within two weeks, are discussed. The cause of diarrhea is believed to be the presence of an organism called Cyanobacteriumlike organism. These are non-refractile, spherical bodies, 8-9 micrometers in diameter, which take on a

IBD-Like Features in Syndromic Diarrhea/Trichohepatoenteric Syndrome.

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Very early onset inflammatory bowel disease (VEOIBD) (inflammatory bowel disease [IBD] before 6 years of age) may manifest as a monogenic disease affecting the gastrointestinal tract. Syndromic diarrhea/trichohepatoenteric syndrome (SD/THE), a rare disorder caused by alteration of a complex involved

Cyanobacteria-like body (CLB) in travellers with diarrhea.

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The cyanobacteria-like body (CLB) is a recently described agent, causing a travellers' disease (TD) syndrome characterized mainly by diarrhea. It has been described in travellers who have visited Asia and Latin America. We report 6 cases seen over a 16-month period among those attending a

Rare AU-1-like G3P[9] human rotaviruses with a Kun-like NSP4 gene detected in children with diarrhea in Italy.

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Three G3P[9] rotaviruses, detected in children hospitalized with gastroenteritis in Palermo, Italy, were found to be genetically related to strains of either human or feline origin in the VP7, VP4, and VP6 genes. In contrast, in the NSP4 gene the viruses resembled G2P[4] human strains, suggesting a

Rotavirus-like infection associated with diarrhea in okapi.

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Effects of human intravenous immune globulin on diarrhea caused by Shiga-like toxin I and Shiga-like toxin II in infant rabbits.

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Shiga toxin and the related Shiga-like toxins (SLT), produced by Escherichia coli, can cause hemorrhagic colitis and hemolytic uremic syndrome (HUS). Human intravenous immune globulin (HIVIg) blocks the cytotoxicity of some SLTs in vitro. To examine the ability of HIVIg to modify disease caused by

Bovine Viral Diarrhea Virus Type 2 Impairs Macrophage Responsiveness to Toll-Like Receptor Ligation with the Exception of Toll-Like Receptor 7.

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Bovine viral diarrhea virus (BVDV) is a member of the Flaviviridae family. BVDV isolates are classified into two biotypes based on the development of cytopathic (cp) or non-cytopathic (ncp) effects in epithelial cell culture. BVDV isolates are further separated into species, BVDV1 and 2, based on

Parvovirus-like particles associated with diarrhea in unweaned piglets.

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Numerous parvovirus-like particles, 18 to 26 nm in diameter, were detected by electron microscopy in the intestinal contents of two to three week old piglets with mild to severe diarrhea, in six Quebec pig herds. Hemagglutination of guinea pig and African green monkey red blood cells was obtained

Chinese-like strain of porcine epidemic diarrhea virus, Thailand.

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Since late 2007, several outbreaks of porcine epidemic diarrhea virus (PEDV) infection have emerged in Thailand. Phylogenetic analysis places all Thai PEDV isolates during the outbreaks in the same clade as the Chinese strain JS-2004-2. This new genotype PEDV is prevailing and currently causing

[Etiologic study of 477 cases of cholera-like diarrhea].

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Identification of two antiviral inhibitors targeting 3C-like serine/3C-like protease of porcine reproductive and respiratory syndrome virus and porcine epidemic diarrhea virus.

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Porcine reproductive and respiratory syndrome virus (PRRSV) and porcine epidemic diarrhea virus (PEDV) are highly virulent and contagious porcine pathogens that cause tremendous economic losses to the swine industry worldwide. Currently, there is no effective treatment for PRRSV and PEDV, and

Reovirus-like agent associated with neonatal diarrhea in pronghorn antelope.

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Reovirus-like particles were demonstrated by negative stain electron microscopic examination of the feces from antelope fawns with diarrhea. Fluorescent antibody tests on frozen sections of ileum from one dead antelope fawn and immunoelectron microscopy tests on feces from two live fawns provided

Reovirus-like agent associated with fatal diarrhea in neonatal pigs.

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Large numbers of a reovirus-like agent were visualized with electron microscopy in bacteria-free gut homogenates obtained from piglets with a fatal diarrhea resembling transmissible gastroenteritis. The syndrome, of vomiting, diarrhea, dehydration, and death, was reproduced in piglets artificially

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