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triolein/hepatitis

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[Absorption of 131-I labeled triolein during viral hepatitis].

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[Exploration of the lipolytic function of the pancreas, using triolein-I-131, in acute viral hepatitis].

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Relationship of phagocytic activity to pathogenicity of mouse hepatitis virus as affected by triolein and cortisone.

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[Studies of the exocrine function of the pancreas in chronic hepatitis, using I-131 labeled triolein and scintigraphy].

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Use of the triolein breath test in alcoholics with liver damage.

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The triolein breath test (TBT) is a simple, noninvasive technique for the evaluation of steatorrhea. However, because it depends on intermediary hepatic processes, and because both liver damage and pancreatic dysfunction often co-exist in the alcoholic, the overall usefulness of the test in patients

Intestinal lipoproteins in the rat with D-(+)-galactosamine hepatitis.

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D-(+)-galactosamine (GalN) induces severe reversible hepatocellular injury in the rat accompanied by lecithin: cholesterol acyltransferase (LCAT) deficiency, defective chylomicron (CM) catabolism, and accumulation of abnormal plasma lipoproteins (Lps), including discoidal high density lipoproteins
OBJECTIVE Dietary intake of the natural omega-3 fatty acid docosahexaenoic acid (DHA) has been implicated in protecting patients with viral hepatitis B or C from developing hepatocellular carcinoma (HCC). Little is known about the effects of DHA on established solid tumors. Here we describe a

PPARA agonist fenofibrate enhances fatty acid β-oxidation and attenuates polycystic kidney and liver disease in mice.

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Peroxisome proliferator-activated receptor α (PPARα) is a nuclear hormone receptor that promotes fatty acid β-oxidation (FAO) and oxidative phosphorylation (OXPHOS). We and others have recently shown that PPARα and its target genes are downregulated, and FAO and OXPHOS are impaired in autosomal
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