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antioxidant/pendarahan
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The effects of antioxidants on a porcine model of liver hemorrhage.
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The aim of this study is to assess the efficacy of the combination of N-acetylcysteine (NAC) and deferoxamine (DFO) in the resuscitation from hemorrhagic shock in a porcine model of bleeding during hepatectomy.
Twenty-one pigs were divided randomly to three groups: Sham (S) group, n = 5; fluid (F)
[Influence of occupational activity on serum vitamin antioxidants content in patients after hemorrhagic fever with renal syndrome].
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Studies covered changes in serum vitamin antioxidant content in patients with hemorrhagic fever with renal syndrome male workers, in accordance with occupational activity type. Findings are that during the disease and convalescence period the drivers examined demonstrated reliably lower levels of
[Impact of blood loss on lipid peroxidation and on the antioxidative system in victims with isolated injury to the chest and abdomen].
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The paper deals with the impact of the volume of blood loss on the time course of changes in the parameters of lipid peroxidation (LPO) and the antioxidative system (AOS) in the sera of 22 and 31 victims with isolated chest and abdominal injuries, respectively, on days 1, 3, 7, and 14 days after
Effect of dietary protein and selected antioxidants on fatty liver hemorrhagic syndrome induced in Japanese quail.
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The effects of CP and antioxidants on fatty liver hemorrhagic syndrome (FLHS) in Japanese quail hens were studied. In Experiment 1, four treatments were arranged as a 2 x 2 factorial; dietary CP (18 or 24%) and reduced glutathione (GSH, 0 or 120 mg/kg diet) were the major variables, but cysteine and
[Effect of the histamine H2-receptor antagonist zantac on lipid peroxidation an antioxidant system in patients with gastroduodenal hemorrhage of ulcer etiology].
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High lipid peroxidation rates and lower antioxidative protection were found in the serum of 64 patients with gastric (n = 20) and duodenal (n = 44) ulcers complicated by hemorrhage. The administration of zantac, a histamine H2-receptor blocker, considerably reduced lipid peroxidation and enhanced
Dietary antioxidant intake and risk of an aneurysmal rupture subarachnoid hemorrhage in Japan.
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The purpose of this study was to investigate the relationship between dietary antioxidant intake and the risk of subarachnoid hemorrhage (SAH) in a case-control study. An antioxidant intake score was calculated for individual values for each food group (soy products, green yellow-vegetables, fruits,
Picroliv modulates antioxidant status and down-regulates AP1 transcription factor after hemorrhage and resuscitation.
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Resuscitation from hemorrhagic shock initiates profound changes in the liver that are likely to contribute to end organ damage and resultant dysfunction after shock. Extensive research in this area has indicated the potential of free radical scavenging strategy for better management of the
Obestatin alleviates subarachnoid haemorrhage-induced oxidative injury in rats via its anti-apoptotic and antioxidant effects.
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OBJECTIVE
The aim was to investigate the putative anti-inflammatory and anti-apoptotic effect of obestatin in a rat model of subarachnoidal haemorrhage (SAH).
METHODS
To induce SAH, rats were injected with 0.3 mL blood into their cisterna magna. At 48 hours rats were decapitated after neurological
Role of acid back-diffusion, glutathione, oxyradical, and histamine in antral hemorrhagic ulcer in rats: the protective effect of lysozyme chloride and antioxidants.
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The pathogenesis of gastric antral hemorrhage and ulceration is unclear. This paper first proposes that antral hemorrhagic ulcers produced in rats are associated with attenuation of defensive parameters (such as mucosal glutathione levels and histamine release, as well as aggravation of aggressive
Constituents and Antioxidant Activity of Bleeding Sap from Various Xinjiang Grapes.
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UNASSIGNED
Wine grape sap or bleeding sap of grapes (GBS) is commonly used in Xinjiang (China) for therapeutic aims. Do variations in composition related to region and variety affect its properties?
UNASSIGNED
GBS samples originating in various parts of Xinjiang (Turpan, Hotan, Kashgar, and Atush)
[Inhibition of lipid peroxidation by antioxidants in the vitreous body during hemorrhage].
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The paper was to investigate the character of malonic dialdehyde content shifts in the vitreous body under the effect of potassium fenosane, oxipyridinchlorhydrate (OPChH), superoxidedismutase, sodium diethylditiokarbamat (DDTK), forming complex with copper under the experimental hemorrhage.
Neuroprotective potential of glibenclamide is mediated by antioxidant and anti-apoptotic pathways in intracerebral hemorrhage.
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The sulfonylurea receptor 1 (SUR1)-regulated NCca-ATP channels were progressively upregulated and demonstrated unchecked opening in central nervous system (CNS) injury, which induced cerebral damage. Glibenclamide (GLI) can block NCca-ATP channels and consequently exert protective effects. Recent
Early antioxidant therapy with Tempol during hemorrhagic shock increases survival in rats.
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BACKGROUND
Hemorrhagic shock (HS) is associated with the generation of reactive oxygen species, which may contribute to delayed multiple organ system failure and death. Previous studies have shown that the antioxidant Tempol improved physiologic variables, although not necessarily outcome, in septic
The joint effect of oxidative stress and antioxidants on the risk of an aneurysmal rupture subarachnoid hemorrhage: a case-control study in Japan.
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OBJECTIVE
A case-control study was conducted in Japan to examine the joint effects of oxidative stress and antioxidants on the risk of subarachnoid hemorrhage (SAH).
METHODS
The study included 201 consecutive patients with first spontaneous SAH aged 30 to 79 years who had aneurysm(s) confirmed by
Combined argatroban and anti-oxidative agents prevents increased vascular contractility to thrombin and other ligands after subarachnoid haemorrhage.
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OBJECTIVE
Increased vascular contractility plays a fundamental role in cerebral vasospasm in subarachnoid haemorrhage (SAH). We investigated the role of thrombin and its receptor, proteinase-activated receptor 1 (PAR1), and other G protein-coupled receptors in the increased contractility, and
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