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aortic valve stenosis/triglyceride
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Triglycerides and remnant cholesterol associated with risk of aortic valve stenosis: Mendelian randomization in the Copenhagen General Population Study.
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[Smoking, increase in plasma lipoprotein (a) and triglycerides, as well as decrease in plasma HDL-cholesterol concentrations seem to be linked with aortic valve stenosis and its progression].
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OBJECTIVE
Evaluation of the plasma levels of total cholesterol, LDL-cholesterol, HDL-cholesterol, triglicerides, lipoprotein (a) during 12 months of the natural course of AVS.
METHODS
60 patients with AVS and 14 controls had echocardiographic examination at the beginning and at the 12th months of
Triglyceride-rich particles: new actors in valvular aortic stenosis
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Administration of angiotensin-converting enzyme inhibitors is associated with slow progression of mild aortic stenosis in Japanese patients.
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It is almost unknown which demographic factors or medications affect the progression of aortic stenosis (AS) in Japanese patients with mild AS. We identified a total of 194 patients with native tricuspid valvular AS, defined as a continuous-wave Doppler determined peak aortic valve jet velocity of ≥
Design and baseline characteristics of the simvastatin and ezetimibe in aortic stenosis (SEAS) study.
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Aortic valve stenosis and atherosclerotic disease have several risk factors in common, in particular, hypercholesterolemia. Histologically, the diseased valves appear to have areas of inflammation much like atherosclerotic plaques. The effect of lipid-lowering therapy on the progression of aortic
Carotid plaque, intima-media thickness, and incident aortic stenosis: a prospective cohort study.
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OBJECTIVE
Aortic stenosis (AS) shares risk factors with atherosclerotic vascular disease. Carotid intima-media thickness (IMT) and plaque may reflect the cumulative damage from exposure to different atherosclerotic risk factors. We examined the relationship of carotid IMT and plaque with incident AS
Can serum lipid and CRP levels predict the "severity" of aortic valve stenosis?
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BACKGROUND
Results of the studies performed have suggested that hypercholesterolaemia and inflammation are important aetiologic factors in aortic valve stenosis (AVS). However up to now no such data has been obtained to evaluate whether these predictors may still serve as valuable tools to estimate
The relation between homocysteine and calcific aortic valve stenosis.
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BACKGROUND
In patients diagnosed with calcific aortic valve stenosis, cardiac risk factors are similar to those of coronary artery disease; homocysteine concentration is an independent risk factor for coronary artery disease. The aim of this study was to investigate the correlation between plasma
Plasma lipoprotein(a) concentration as an independent predictor of hemodynamic progression of aortic valve stenosis
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Calcific aortic valve disease (CAVD) is a common cardiovascular disorder of high social significance. This study aimed to identify independent predictors of hemodynamic progression of CAVD. The relationship between some risk factors, including the rs10455872 polymorphism in the intron 25 of the
Apolipoproteins AI, B, and E polymorphisms in severe aortic valve stenosis.
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Hypercholesterolemia has been related to aortic valve stenosis (AS). Polymorphisms of apolipoproteins (apo) AI, B, and E are associated with variable levels of plasma lipids, but the association between these polymorphisms and AS is unknown. In a case-control study of groups matched by age, sex,
Increased insulin, triglycerides, reactive oxygen species, and cardiac fibrosis in mice with a mutation in the helicase domain of the Werner syndrome gene homologue.
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Werner Syndrome (WS) is a rare disorder characterized by the premature onset of a number of age-related diseases. The gene responsible for WS encodes a DNA helicase/exonuclease protein. Previously, we generated a mouse model lacking part of the helicase domain of the murine Wrn homologue. Mutant
Myocardial steatosis and left ventricular contractile dysfunction in patients with severe aortic stenosis.
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BACKGROUND
Aortic stenosis (AS) leads to left ventricular (LV) hypertrophy and dysfunction. We hypothesized that cardiac steatosis is involved in the pathophysiology and also assessed whether it is reversible after aortic valve replacement.
RESULTS
Thirty-nine patients with severe AS
[Aortic stenosis and coronary disease. Analysis of risk factors].
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OBJECTIVE
To analyze clinical laboratorial aspects of the presence of coronary disease in patients with aortic stenosis and evaluate the influence of risk factors in the development of obstructive coronary disease.
METHODS
We studied 65 patients who had severe aortic stenosis with an indication for
Atorvastatin therapy is not associated with slowing the progression of aortic stenosis: findings of a randomized controlled trial.
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BACKGROUND
Aortic stenosis (AS) is the most common type of valvular cardiac disorders. AS has many risk factors in common with atherosclerosis. Hypercholesterolemia is an important pathomechanism for AS. However, the impact of statin drugs on slowing AS progression has not yet been well
[Body mass index influence on aortic valve stenosis].
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OBJECTIVE
The 12 months' observation of body mass index (BMI) influence on natural course of aortic valve stenosis (AVS).
METHODS
60 AVS patients who did not agree for operational treatment were divided into group A (n = 15) with BMI 20-25, group B (n = 27) with BMI 25,01-30 and group C BMI >
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