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Demonstration of viral inclusion bodies in unstained tissue sections with the aid of the phase microscope; the inclusion bodies of yellow fever, herpes simplex, fowl pox, and distemper.
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DEMONSTRATION OF VIRAL INCLUSION BODIES IN UNSTAINED TISSUE SECTIONS WITH THE AID OF THE PHASE MICROSCOPE I. : The Inclusion Bodies of Yellow Fever, Herpes Simplex, Fowl Pox, and Distemper.
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[Classical article. Canine distemper (canine epizootic catarrhal fever). By J.C. van der Slooten. 1894].
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Canine distemper virus epithelial cell infection is required for clinical disease but not for immunosuppression.
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To characterize the importance of infection of epithelial cells for morbillivirus pathogenesis, we took advantage of the severe disease caused by canine distemper virus (CDV) in ferrets. To obtain a CDV that was unable to enter epithelial cells but retained the ability to enter immune cells, we
Canine distemper virus infection in fennec fox (Vulpes zerda).
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Fifteen 8-month-old fennec foxes imported from Sudan showed fever, mucopurulent ocular discharge, diarrhea, severe emaciation, seizures, and generalized ataxia, and died. Three of the 15 animals were presented for diagnostic investigation. Severe dehydration, brain congestion, and gastric ulcers
Canine distemper virus (CDV) infection of ferrets as a model for testing Morbillivirus vaccine strategies: NYVAC- and ALVAC-based CDV recombinants protect against symptomatic infection.
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Canine distemper virus (CDV) infection of ferrets causes an acute systemic disease involving multiple organ systems, including the respiratory tract, lymphoid system, and central nervous system (CNS). We have tested candidate CDV vaccines incorporating the fusion (F) and hemagglutinin (HA) proteins
EVIDENCE OF TWO COCIRCULATING CANINE DISTEMPER VIRUS STRAINS IN MESOCARNIVORES FROM NORTHERN COLORADO, USA.
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Canine distemper virus (CDV) is a highly contagious pathogen that principally infects wildlife and domestic carnivores. Peridomestic species such as raccoons ( Procyon lotor) experience outbreaks with high mortality. Clinical signs of infection include anorexia, fever, respiratory infection, and
Humoral immunity to canine distemper after immunization of dogs with inactivated and live measles virus.
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Pairs of dogs were immunized with 2 injections of a) measles virus material containing hemagglutinin but no hemolysin [Tween 80 and ether (TE) treated] b) material containing both hemagglutinin and hemolysin (freeze-dried and heated at 95 degrees C for 30 minutes), and c) live measles virus. Two
Pathogenesis of canine distemper virus in experimentally infected raccoon dogs, foxes, and minks.
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Canine distemper virus (CDV) infects a broad range of carnivores and causes a highly contagious disease with severe immunosuppression. The disease severity markedly varies in different species. To investigate the pathogenesis of CDV in raccoon dog (Nyctereutes procyonoides), fox (Vulpes vulpes) and
CANINE DISTEMPER IN A VACCINATED SNOW LEOPARD ( PANTHERA UNCIA).
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A 6-yr-old male snow leopard ( Panthera uncia) presented with acute seizures, hyperthermia, and tachypnea. Because of a diagnosis of anuric renal failure, the animal was euthanized. On histopathologic examination, numerous intralesional intracytoplasmic and intranuclear inclusions were found in the
Comparison of tissue and fluid samples for the early detection of canine distemper virus in experimentally infected dogs.
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The clinical utility of various specimens was examined for the early diagnosis of canine distemper (CD). Seven healthy dogs at 17 weeks of age were experimentally infected with a field isolate of canine distemper virus. The RT-PCR was carried out to detect CDV NP gene. Dogs showed mild fever and
Canine distemper and related diseases: report of a severe outbreak in a kennel.
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An outbreak of canine distemper in a kennel of German shepherds in the province of Bari is reported. Six 42-day-old pups developed typical signs of canine distemper (fever, conjunctivitis, respiratory distress and enteritis) and died within 7-10 days. Neurological symptoms were observed only in one
Measles vaccination of nonhuman primates provides partial protection against infection with canine distemper virus.
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Measles virus (MV) is being considered for global eradication, which would likely reduce compliance with MV vaccination. As a result, children will grow up without MV-specific immunity, creating a potential niche for closely related animal morbilliviruses such as canine distemper virus (CDV).
Delineating morbillivirus entry, dissemination and airborne transmission by studying in vivo competition of multicolor canine distemper viruses in ferrets.
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Identification of cellular receptors and characterization of viral tropism in animal models have vastly improved our understanding of morbillivirus pathogenesis. However, specific aspects of viral entry, dissemination and transmission remain difficult to recapitulate in animal models. Here, we used
Outbreak off canine distemper in vaccinated dogs in Finland.
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Canine distemper reappeared in dogs in Finland in 1990 after a 16-year absence. In 1994 to 1995 an outbreak occurred in areas with a high density dog population which involved dogs vaccinated against distemper. The estimated total number of cases was at least 5000, and 865 cases were confirmed by
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