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hypocapnia/infark
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Regional cerebral blood flow response to hypocapnia in the contralateral hemisphere of patients with acute cerebral infarction.
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The rCBF response to hypocapnia induced by active short-term hyperventilation was determined in the contralateral hemispheres of ten patients with acute unilateral cerebral infarction. Overall rCBF reduction occurred in only two patients. Regional or widespread abnormal responses to Paco2 reduction
Effects of hyperventilation, hypocapnia, and acetazolamide on experimental cerebral ischemia and infarction.
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Correlation between the reactivity of cerebral vessels to hypocapnia and the response of the glycostatic mechanism in cerebral infarct patients.
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Effect of hypocarbia and hypercarbia in experimental brain infarction. A microangiographic study in the monkey.
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[Disordered alveolar-capillary gas metabolism in the acute stage of myocardial infarct].
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The alveolar-capillary gas metabolism was studied in 60 patients with acute myocardial infarction. Arterial hypoxemia and hypocapnia were observed in the majority of the patients. Those disturbances are more pronounced in the presence of cardiac insufficiency. The combination of shunt effect with
Aspirin, hyperventilation, and cerebellar infarction in sickle cell disease.
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Aspirin ingestion was followed by hyperventilation, cerebellar signs, and fatal brain stem dysfunction in a patient with sickle cell disease. Autopsy showed a swollen, recently infarcted cerebellum with tonsillar herniation compressing the medulla. We propose that hypocapnea from aspirin-induced
[Controlled hyperventilation in the therapy of acute myocardial infarction].
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Two patients with anterior wall infarction were hyperventilated under control with the aim of the induction of an inverse steal-phenomenon. Simultaneously the registration of the ST-dynamics was performed by means of precordial mapping. In a region of an arterial carbon dioxide partial pressure up
[Blood gas analysis and acid-base balance in acute myocardial infarction. Personal observations (author's transl)].
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Arterial pH, pO2 and pCO2 were analysed with Astup's micromethod on one hundred and three acute myocardial infarctions (A.M.I.) without metabolic, pulmonary and renal diseases. Following the clinical picute, the patients were divided into five groups and results were clinically and statistically
Improvement of cerebral blood flow and/or CO2 reactivity after superficial temporal artery-middle cerebral artery bypass in patients with transient ischemic attacks and watershed-zone infarctions.
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The effect of extracranial-intracranial bypass anastomosis on cerebral blood flow and CO2 reactivity during hypocapnia was investigated in ten patients with transient ischemic attacks or watershed infarctions due to carotid occlusive diseases. Six patients had occlusion and four had stenosis
Vascular response to carbon dioxide in areas with and without diaschisis in patients with small, deep hemispheric infarction.
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The reactivity of cerebral blood vessels to changes in PaCO2 in areas of the cerebral cortex with or without diaschisis was investigated in 13 patients in a subacute or chronic stage after a small capsular infarct. A focal area of hypoperfusion (area of diaschisis) was detected in the ipsilateral
Failure of prolonged hypocapnia, hypothermia, or hypertension to favorably alter acute stroke in primates.
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The effects of induced hypocapnia, hypothermia, and hypertension were surveyed in a primate model of acute stroke during and following a 48-hour period of intensive care. The results were compared to a group of nine control animals previously studied. Hypocapnia (PaCO2=25 torr) was examined in five
Circulatory reflexes in myocardial infarction.
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The Valsalva manoeuvre was used to examine circulatory reflexes in 22 patients with acute myocardial infarction. Four patients had the 'square wave' response of cardiac failure and two responded normally to the manoeuvre. In the remaining 18 patients there was a 20 per cent or more fall in pulse
Posttraumatic cerebral infarction in patients with moderate or severe head trauma.
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OBJECTIVE
To evaluate the frequency, types, and location of posttraumatic cerebral infarction, to assess if secondary cerebral insults were associated with cerebral infarction, and to determine if cerebral infarction affected patients' outcome.
METHODS
We based diagnosis of cerebral infarction on
Vasoreactivity and peri-infarct hyperintensities in stroke.
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OBJECTIVE
It is unknown if impaired cerebral vasoreactivity recovers after ischemic stroke, and whether it compromises perfusion in regions surrounding infarct and other vascular territories. We investigated the regional differences in CO2 vasoreactivity (CO2 VR) and their relationships to
[Vasoreactivity of cerebral infarct to barbiturates and other intravenous anesthetics].
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The effect of a barbiturate, Sodium thiopental (Nesdonal), on the cerebral circulation in 13 patients with a cerebral ischemic attack is studied. Its action is compared to the action of other intravenous anesthetics (Alfatésine - Etomidate - diazanalgésie) used in neuro-anesthésia. The study is on a
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