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hypocapnia/sembap
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Hypocapnia worsens arterial blood oxygenation and increases VA/Q heterogeneity in canine pulmonary edema.
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BACKGROUND
Hyperventilation frequently is employed to reduce carbon dioxide partial pressure in patients in the operating room and intensive care unit. However the effect of hypocapnia on oxygenation is complex and may result in worsening in patients with preexisting intrapulmonary shunt. To better
Hypocapnia in acute pulmonary edema.
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Role of hypocapnia in the alveolar surfactant increase induced by free fatty acid intravenous infusion in the rabbit.
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Intravenous infusion of free fatty acid (FFA) produces an increase in the alveolar surfactant pool of the rabbit and pulmonary edema, hyperventilation, hypoxemia and hypocapnia. Previous studies suggested that alveolar PCO2 would be a regulator of intracellular storages of surfactant. In order to
Non-invasive pressure support ventilation in patients with respiratory failure due to severe acute cardiogenic pulmonary edema.
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BACKGROUND
Recent studies suggest the use of non-invasive pressure support ventilation (NIPSV) in patients with acute cardiogenic pulmonary edema (ACPE). However, it remains unclear whether all patients with ACPE benefit from NIPSV.
OBJECTIVE
To investigate short-term effects of NIPSV on
Brain cell swelling during hypocapnia increases with hyperglycemia or ketosis.
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BACKGROUND
Severe hypocapnia reduces cerebral blood flow (CBF) and is known to be a risk factor for diabetic ketoacidosis (DKA)-related cerebral edema and cerebral injury in children. Reductions in CBF resulting from hypocapnia alone, however, would not be expected to cause substantial cerebral
The effect of hypocapnia and mechanical pulmonary stress on lung tissue in newborn lambs.
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Positive pressure ventilation, using high inspiratory pressures, often causes lung damage. When associated with hypocapnia, it can produce severe focal alveolar alkalosis and can cause damage in areas of low blood flow. A vein-to-vein extracorporeal membrane oxygenator (ECMO) system was used to
Early Hours in the Development of High Altitude Pulmonary Edema- Time Course and Mechanisms.
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Clinically evident high altitude pulmonary edema (HAPE) is one of severe cyanosis, dyspnea and edema. This usually occurs within 1-2 days of ascent often with additional stresses of exercise and sleep-related hypoventilation. The earliest events in HAPE occur rapidly and progress through clinically
Mortality in acute cardiogenic pulmonary edema treated with continuous positive airway pressure.
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OBJECTIVE
To investigate mortality in acute cardiogenic pulmonary edema (ACPE) patients treated with continuous positive airway pressure (CPAP) and to identify clinical and laboratory characteristics associated with mortality.
METHODS
Observational, retrospective study.
METHODS
Emergency Medicine
[Current status of treatment of the cerebral edema].
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The treatment of cerebral edema has changed during recent years. On the one hand, knowledge of the pathophysiology of brain swelling has expanded; on the other, the analysis of biodata such as intracranial pressure, cerebral blood flow, and blood volume has become routine. The methods of measuring
Post-anesthetic pulmonary edema in two horses.
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CASE 1: A two-year old, 462 kg Standard bred horse was anesthetized for arthroscopy and castration. During anesthesia, hyperemia of the mucosal membranes and urticaria were noticed. During 5 hours of anesthesia subcutaneous edema of the eyelids and neck region developed. In the recovery box, the
[Effect of hypocapnia/alkalosis on the fluid filtration rate in isolated and perfused rabbit lungs].
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Hypocapnia/alkalosis is a consequence of several lung and metabolic pathologies. The aim of this study was to determine whether the increase of fluid filtration rate (FFR) that occurs during Hypocapnia/alkalosis circumstances is determined by hypocapnia, alkalosis or both. 7 groups were formed
Cerebral vasoconstriction in response to hypocapnia is maintained after ischemia/reperfusion injury in newborn pigs.
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OBJECTIVE
Hypocapnic cerebral vasoconstriction is used therapeutically to reduce elevated intracranial pressure caused by cerebral edema. Because cerebral ischemia/reperfusion injury causes a selective loss of prostanoid-dependent responses, including vasodilation to hypercapnia, we designed these
Failure of prolonged hypocapnia, hypothermia, or hypertension to favorably alter acute stroke in primates.
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The effects of induced hypocapnia, hypothermia, and hypertension were surveyed in a primate model of acute stroke during and following a 48-hour period of intensive care. The results were compared to a group of nine control animals previously studied. Hypocapnia (PaCO2=25 torr) was examined in five
Risk factors for intubation as a guide for noninvasive ventilation in patients with severe acute cardiogenic pulmonary edema.
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OBJECTIVE
Noninvasive ventilation may reduce the endotracheal intubation rate in patients with acute cardiogenic pulmonary edema. However, criteria for selecting candidates for this technique are not well established. We analyzed a cohort of patients with severe acute cardiogenic pulmonary edema
Prevalence of cerebral edema among diabetic ketoacidosis patients
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Introduction: Cerebral edema (CE) is the most severe complication of diabetic ketoacidosis (DKA) in children. There is no accurate knowledge of CE pathogenesis and its onset has been related to intravenous rehydration therapy during the
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