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lithocarpus hancei/antijamur
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Sweet tea leaves extract improves leptin resistance in diet-induced obese rats.
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OBJECTIVE
Dietary obesity is usually characterized by leptin resistance and abnormal lipid metabolism. Lithocarpus polystachyus Rehd.(Sweet Tea) leaf is a kind of Chinese folkloric medicine, and it has been widely used for obesity, diabetes, and hypertension in South China. The present study is
An exploratory study of the nutritional composition of Tanoak (Lithocarpus densiflorus) acorns after potassium phosphonate treatment.
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Native American Pomo communities who live in the Northern Coastal range of California and consume acorns from tanoak trees as part of traditional diets are facing the potential loss of many culturally important trees to sudden oak death. Pomo and other Native American communities are reluctant to
Neuroprotective effects of trilobatin, a novel naturally-occurring Sirt3 agonist from Lithocarpus polystachyus Rehd., mitigates cerebral ischemia/reperfusion injury: Involvement of TLR4/NF-κB and Nrf2/ Keap-1 signaling.
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Lithocaldehydes A and B, polyketones from the deep sea-derived fungus Phomopsis lithocarpus FS508
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Lithocaldehydes A (1) and B (2), a pair of novel diastereoisomers possessing an unprecedented 6/6/5/5/6 highly-fused ring system forming an earring-like skeleton, were isolated from the deep sea-derived fungus Phomopsis lithocarpus FS508. The structures of 1 and 2 were fully characterized and
Phytopthora ramorum in Belgium: 2002 survey results and research efforts.
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Phytophthora ramorum is a new and aggressive Phytophthora species that causes leaf blight and dieback symptoms on Viburnum and Rhododendron plants in Europe. A variant of this fungus is responsible for Sudden Oak Death (SOD) in California and Oregon. In Europe, problems so far are mostly restricted
Trilobatin Protects Against Aβ 25-35-Induced Hippocampal HT22 Cells Apoptosis Through Mediating ROS/p38/Caspase 3-Dependent Pathway
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Emerging evidence reveals that an aberrant accumulation of β-amyloid (Aβ) is the main reason of Alzheimer's disease (AD) pathogenesis. Thus, inhibition of Aβ-induced neurotoxicity may be promising therapeutic tactics to mitigate AD onset and advance. The development of agent candidates by cultured
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