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muscle hypotonia/hypoxia
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[Respiratory muscle hypotonia as one of the causes of hypoventilation and hypoxemia in patients with chronic renal failure treated by hemodialysis].
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In 21 patients with chronic renal failure spirometric and electromyographic (included m. pectoralis, m. rectus abdomini, m. obligus abdomini) examinations were performed before and after hemodialysis. We observed a decreased myoelectrical activity of muscles on the basis of amplitude of EMG.
Susceptibility of the cat's visual system to hypoxia, hypotonia and circulatory arrest.
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[Regulation of kidney function in exsiccosis; the role of cerebral hypotonia, hypoxemia and hypercapnia in the regulation of renal circulation].
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Characterization of pontine neurons which respond to hypoxia in fetal sheep.
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Hypoxia causes apnea and postural muscle hypotonia in fetal sheep, a response thought to arise by descending inhibition from a group of lateral pontine neurons that express FOS protein after hypoxia. To determine the neurochemical phenotype, and whether these neurons project to the cervical spinal
[Perinatal hypoxia at Hospital Mederi in Bogotá: behavior in the period 2007-2011].
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UNASSIGNED
To identify maternal, fetal and neonatal risk factors associated with perinatal hypoxia in neonates at Hospital Universitario Mayor Mederi (HUM Mederi) from 2007 to 2011.
UNASSIGNED
In the period 2007-2011, 8 837 children were born in HUM Mederi. Only the cases that presented with
[Adaptational disorders of the cardiovascular regulation during brief exposure to hypobaric hypoxia].
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Responses of the cardiovascular system to brief exposure to hypobaric hypoxia were the focus of this investigation. Impairment of adaptive reactions of the body was found to depend on the level of physical loading. "Critical" time of exposure to a simulated altitude of 5000 m came to 7-10 minutes.
Effect of transitory ischemia on liver lysosomal apparatus in rats with different resistance to hypoxia.
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We studied the state of lysosomal apparatus and pro- and antioxidant activity in the liver of rats with different resistance to hypoxia during postischemic recovery. Under normal conditions the lysosomal apparatus did not differ in highly and low resistant animals. During ischemia and reperfusion
Near-term fetal hypoxia-ischemia in rabbits: MRI can predict muscle tone abnormalities and deep brain injury.
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OBJECTIVE
The pattern of antenatal brain injury varies with gestational age at the time of insult. Deep brain nuclei are often injured at older gestational ages. Having previously shown postnatal hypertonia after preterm fetal rabbit hypoxia-ischemia, the objective of this study was to investigate
Elevated spinal monoamine neurotransmitters after antenatal hypoxia-ischemia in rabbit cerebral palsy model.
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We hypothesized that a deficiency in the descending serotonergic input to spinal cord may underlie postnatal muscle hypertonia after global antenatal hypoxic-ischemic injury in a rabbit model of cerebral palsy. Neurotransmitter content was determined by HPLC in the spinal cord of newborns with and
Restructuring of sleep and reversal of REM-induced supraspinal hypotonia of respiratory muscles following bilateral phrenicotomy.
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The long-term effect of diaphragm paralysis on respiratory system function is still not clear. We monitored changes in breathing pattern and the sleep/wake cycle in a dog before and after bilateral phrenicotomy. The post-operative observation extended over 6 months. It was noted that minute
Congenital central alveolar hypoventilation syndrome (Ondine's curse) with survival into adulthood.
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Ondine's curse or central alveolar hypoventilation (CCAH) syndrome is a disorder of the autonomic nervous system resulting in respiratory dysregulation. The clinical outcome is typically poor, with few individuals living into adulthood and even fewer surviving to adulthood with normal neurological
Motor control of the pharyngeal musculature and implications for the pathogenesis of obstructive sleep apnea.
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Obstructive sleep apnea is a common breathing problem that results in recurrent episodes of nighttime hypoxemia, hypercapnia, bradytachycardia, and hypertension, as well as sleep disturbance and daytime hypersomnolence. The obstruction is located in the oropharynx and is caused by hypotonia of the
The mechanism of dead-in-bed syndrome and other sudden unexplained nocturnal deaths.
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The mechanism of dead-in-bed syndrome (DBS), a rare but devastating condition that mainly affects young type 1 diabetes patients, remains mysterious. A new theory is proposed to explain this syndrome. This theory suggests that repeated episodes of hypoglycaemia-induced adaptation in orexin-A neurons
[Obstructive sleep apnea syndrome in a child with trisomy 21].
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Pulmonary hypertension without any cardiovascular malformation was diagnosed by heart catheterization in a 4 year old girl with trisomy 21. A suspected obstructive sleep apnea syndrome was confirmed by polysomnography which revealed numerous obstructive apneas and hypopneas (apnea-index 23/h) with
Neonatal myasthenia gravis: specific advantages of repetitive stimulation over edrophonium testing.
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A premature infant with neonatal myasthenia gravis is presented to illustrate the utility of electrodiagnosis. The patient, born to a mother with myasthenia gravis, suffered additional problems, including hypoxia and subependymal hemorrhage which potentially contributed to hypotonia and poor
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