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nesidioblastosis/obesitas
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B-cell nesidioblastosis in the overweight newborn.
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The endocrine pancreata of five overweight newborns (birthweight ranging from 3,800 g to 4,500 g) have been studied. Basal blood glucose and insulin levels were normal in these newborns. A marked and widespread B-cell nesidioblastosis was observed in relation to ten controls (birthweight ranging
One-Anastomosis Jejunal Interposition with Gastric Remnant Resection (Branco-Zorron Switch) for Severe Recurrent Hyperinsulinemic Hypoglycemia after Gastric Bypass for Morbid Obesity.
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The anatomical and physiological changes after Roux-en-Y gastric bypass for morbid obesity can lead to severe hyperinsulinemic hypoglycemia with neuroglycopenia in a small percentage of patients. The exact physiologic mechanism is not completely understood. Surgical reversal to the original anatomy
Functional imaging in hyperinsulinemic hypoglycemia after gastric bypass surgery for morbid obesity.
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BACKGROUND
Hyperinsulinemic hypoglycemia after Roux-en-Y gastric bypass (RYGB) has been increasingly reported. It is induced by β-cell hyperplasia often referred to as nesidioblastosis. Positron emission tomography (PET) with [11C]-5-hydroxytryptophan ((11)C-HTP) and
Restoration of insulin sensitivity in the obese mouse following suppression of pancreatic islet cell hyperplasia.
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The pathogenesis of pancreatic islet cell hyperplasia and insulin insensitivity in obesity.
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Adult diffuse nesidioblastosis: genetically or environmentally induced?
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Persistent hyperinsulinemic hypoglycemia in adults is usually caused by solitary benign insulinomas. Nesidioblastosis, a term that has been used to designate a functional disorder of the beta cells, is a rare cause of persistent hyperinsulinemic hypoglycemia in adults, but seems to have increased in
A rare case of non-insulinoma pancreatic hypoglycaemia syndrome (niphs) in an adult due to localised islet cell hyperplasia-successfully managed by enucleation.
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Persistent hyperinsulinemic hypoglycaemia is caused most commonly by an insulinoma in adults or by nesidioblastosis in neonates. In adults, localised islet cell hyperplasia is a rare disorder characterised by localised proliferation of islet cells. The authors present the case of a previously
Reversible hyperinsulinemic hypoglycemia after gastric bypass: a consequence of altered nutrient delivery.
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BACKGROUND
Severe hypoglycemia after Roux-en-Y gastric bypass surgery (RYGB) is an increasingly recognized condition, characterized by neuroglycopenia and inappropriately elevated insulin concentrations that occur primarily in the postprandial state. Both pathophysiology and treatment of this
Did the gradual loss of GLUT2 cause a shift to diabetic disorders in the New Zealand obese mouse (NZO/Hl)?
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The New Zealand obese mouse (NZO/Hl) is characterised by hereditary obesity and type-2 diabetes, including insulin resistance, hyperinsulinaemia, and glucose intolerance. In other diabetic models, it has been revealed that the proper functioning of the glucose transporter isoform 2 (GLUT2) is
Hyperinsulinemic hypoglycemia with nesidioblastosis after gastric-bypass surgery.
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We describe six patients (five women and one man; median age, 47 years; range, 39 to 54) with postprandial symptoms of neuroglycopenia owing to endogenous hyperinsulinemic hypoglycemia after Roux-en-Y gastric bypass surgery. Except for equivocal evidence in one patient, there was no radiologic
Postprandial hyperinsulinemic hypoglycemia in a child as a late complication of esophageal reconstruction.
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BACKGROUND
Postprandial hyperinsulinemic hypoglycemia (PHH) is an increasingly recognized complication of gastric bypass surgery in obese adults, distinct from the "dumping syndrome".
METHODS
Upon birth, primary repair of esophageal atresia was performed, and at the age of 14 months definite
The relationship between the hyperplastic pancreatic islet and insulin insensitivity in obesity.
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In summary, the present review provides evidence in support of the proposition that pancreatic islet cell hyperplasia precedes the development of insulin insensitivity in the obese mouse and, it is likely, that similar events occur in obese humans. Moreover, the hyperplastic pancreatic islet appears
Successful medical management of status post-Roux-en-Y-gastric-bypass hyperinsulinemic hypoglycemia.
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Roux-en-Y gastric bypass (RYGBP) is the most commonly performed type of bariatric surgery, which is used in the treatment of obesity and type 2 diabetes. Recent case reports and case series have described a rare complication of RYGBP, status post-gastric-bypass hyperinsulinemic hypoglycemia, which
Postprandial hyperinsulinemic hypoglycemia after Roux-en-Y gastric bypass: an update.
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Roux-en-Y gastric bypass (RYGB) is an efficient treatment for morbid obesity and reduces obesity-related co-morbidities. With the growing number of patients undergoing gastric bypass, complications now demand further attention. Postprandial hyperinsulinemic hypoglycemia (PHH) after Roux-en-Y gastric
A rare complication of gastric bypass (weight loss) surgery: nesidioblastosis.
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Here, we present the case of a 31-year-old woman patient who underwent distal pancreatectomy with the history of gastric bypass surgery for obesity. The final histopathological diagnosis of the lesion was nesidioblastosis. Nesidioblastosis is the most common cause of organic persistent
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