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palmitamide/seizures
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[Effect of glutamic acid palmitamide or linoleamide on convulsions induced by pentetrazole].
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Female Balb-C mice received glutamic acid palmitamide (PGt) or linoleamide (LGt) at doses included between 10 and 135 mg.kg-1 (i.p.) and, 90 min after these injections, pentetrazol (PTZ) at 90 mg.kg-1, i.p. PGt (10 or 100 mg.kg-1, i.p.) reduced significantly latencies of first convulsions and
[Are there glutamatergic, anti-GABAergic or antiglycinergic components in the action mechanism of adamantamines? Study of convulsions induced by amantadine or 1-adamantylcyclopentanamine in mice].
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Convulsions induced on mice by a dose of 200 mg/kg i.p. of amantadine (Ama) or 1-adamantylcyclopentanamine (Ad1) were studied with tricyclic antidepressants (imipramine, desipramine, chlorimipramine), possessing antiglutamatergic properties, or with GABA-linoleamide, a centrally acting GABAergic
Synthesis and anticonvulsant activity of N-(2-hydroxy-ethyl)amide derivatives.
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A series novel of N-(2-hydroxyethyl)amide derivatives was synthesized and screened for their anticonvulsant activities by the maximal electroshock (MES) test, and their neurotoxicity was evaluated by the rotarod test (Tox). The maximal electroshock test showed that N-(2-hydroxyethyl)decanamide 1g,
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