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phytohaemagglutinin/demam
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The in vitro response to phytohaemagglutinin during malignant catarrhal fever of rabbits and cattle.
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Rabbits with malignant catarrhal fever were recently found to have depressed T and B cell function which was shown by failure of their blood lymphocytes to respond to phytohaemagglutinin (PHA) and other mitogens. In the present work, in contrast, lymphocytes from diseased calves were found to
Indomethacin-mediated enhancement of lymphocyte response to phytohaemagglutinin in dengue haemorrhagic fever patients.
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Suppressor cell function in a family with familial Mediterranean fever.
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Defective suppressor cell function has been demonstrated in several diseases but has not been tested in familial Mediterranean fever (FMF). We tested the ability of concanavalin A-activated suppressor cells from one family with FMF to inhibit the proliferation of phytohaemagglutinin-stimulated
Correction of a suppressor cell deficiency in familial Mediterranean fever by colchicine.
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We have previously reported a suppressor cell deficiency in four patients with familial Mediterranean fever (FMF). Since colchicine prevents FMF attacks, we tested the effect of colchicine (1 mg twice daily) on the suppressor cell function in three of these FMF patients. Proliferation of
The effect of culture temperature on the kinetics of human lymphocyte stimulation by phytohaemagglutinin.
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The effect of varying the culture temperature on the growth kinetics of human lymphocytes stimulated by phytohaemagglutinin (PHA) has been studied by a new method using a Coulter Channelyzer. Each of three subjects had a distinctive percentage of responding cells (P less than 0.01), greater after 22
Induced TNF production in vitro as a test for familial Mediterranean fever.
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We have previously demonstrated an altered pattern of tumor necrosis factor (TNF) secretion in patients with familial Mediterranean fever (FMF). To examine whether TNF determination could assist in diagnosing FMF, we stimulated heparinized blood of 51 asymptomatic FMF patients with
Enhancement of IL-1, IL-2 production and IL-2 receptor generation in patients with acute rheumatic fever and active rheumatic heart disease; a prospective study.
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In a prospective study, patients with quiescent rheumatic heart disease (CRHD), streptococcal pharyngitis (SP) and healthy normal subjects produced comparable amounts of IL-1 and IL-2, but acute rheumatic fever (ARF) patients produced significantly elevated amounts of IL-1 and IL-2 at all intervals
Does immune activation continue during an attack-free period in familial Mediterranean fever?
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Although some information is available regarding immune activation in familial Mediterranean fever (FMF), little is known about either peripheral blood T cell activation marker expression or the T cell proliferative response to phytohaemagglutinin (PHA). In the present study, we aimed to investigate
Depression of lymphocyte response to mitogens in sheep infected with tick-borne fever.
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Lymphocytes obtained from sheep experimentally infected with Cytoecetes phagocytophila, the causative agent of tick-borne fever, showed reduced blastogenesis induced by the mitogens phytohaemagglutinin and E. coli lipopolysaccharide. The period of reduced lymphocyte reactivity coincided with the
Immunological effects of in vitro hyperthermia.
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The effect of in vitro hyperthermia treatment on the immunological function of human peripheral blood mononuclear cells (PBMC) was investigated to evaluate the immunosuppressive effect of hyperthermia, which has been successfully used for the treatment of cancer. The viability was slightly reduced
The non-haemadsorbing African swine fever virus isolate ASFV/NH/P68 provides a model for defining the protective anti-virus immune response.
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African swine fever virus ASFV/NH/P68 is a naturally occurring, non-haemadsorbing and non-fatal isolate. Longitudinal clinical and immunological studies on 31 pigs inoculated oronasally or intramuscularly with this isolate defined two discrete groups of animals: those developing ASF chronic type
Immunosuppression in sheep experimentally infected with Ehrlichia phagocytophila.
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The effect of tick-borne fever (TBF) on antibody formation and lymphocyte proliferation in sheep was studied following experimental infection with Ehrlichia phagocytophila. All infected sheep developed fever within three to four days. The sheep recovered clinically within eight days. Both infected
Effect of elevated temperature on human immunoglobulin synthesis, lymphokine production and lymphocyte proliferation in vitro.
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Fever in infections is mediated by endogenous pyrogen, a protein inseparable from interleukin 1 (IL-1). It remains unclear what the role of fever in host defence is. We studied the effect of elevated temperature on mitogen-induced human lymphocyte stimulation in vitro. Lymphocyte proliferation,
[Rapidly debilitating disease with generalized lymphadenopathy, skin involvement and interstitial pulmonary infiltration (report of three cases)].
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Generalized but well-circumscribed lymphadenopathy and rash-like skin changes were observed in three men, aged 58 to 75 years. There was a reticular appearance in the chest X-ray. Dyspnoea, weakness, marked weight loss, changing but marked lymphopenia, markedly increased blood-sedimentation rate,
A cell-free extract of Salmonella typhimurium inhibits mitogen-induced proliferation of murine splenic T lymphocytes.
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In this study, a cell-free extract of Salmonella inhibited T cell mitogen-induced proliferation of spleen cells from non-immunized mice. The proliferation of murine spleen cells stimulated with a T cell mitogen, such as phytohaemagglutinin (PHA) or concanavalin A (ConA) was suppressed significantly
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