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pneumoperitoneum/tyrosine
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The effects of carbon dioxide pneumoperitoneum on tyrosine hydroxylase activity.
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OBJECTIVE
The aim of this study is to investigate the effects of carbon dioxide (CO2) pneumoperitoneum on tyrosine hydroxylase (TH) activity and total protein (TP) levels.
METHODS
Forty male Sprague-Dawley rats were randomized into 10 groups, each consisting of 10 rats. Groups 1 and 2 consisted of
A pneumoperitoneum due to intestinal cystic pneumatosis associated with a tyrosine kinase inhibitor.
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Pneumatosis cystoides intestinalis is characterized by multiple air-filled cysts in the intestine wall. A perforation causes pneumoperitoneum, frequently with minimal clinical affectation and do not require surgery at first in most cases. Recently, some cancer treatments, mainly molecular targeted
[A case of pneumatosis intestinalis associated with sunitinib treatment for renal cell carcinoma].
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Sunitinib as a multitarget tyrosine kinase inhibitor is one of the anti-tumor agents, approved by the United States Food and Drug Administration to use treat gastrointestinal stromal tumor and metastatic renal cell carcinoma. The agent is known to commonly induce adverse reactions such as fatigue,
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