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prosopagnosia/kanker
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[Transient prosopagnosia after removal of a tumor in the right occipito-temporal cortex: a case report].
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A 58-year-old man with metastatic brain tumor in his right occipito-temporal region was operated, using craniectomy. He had no neurological symptoms preoperatively. The tumor was 2.5 cm in diameter with minor perifocal edema. Two days after total removal of the tumor, typical prosopagnosia appeared,
[Transient prosopagnosia and lasting topographical disorientation after the total removal of a right occipital arteriovenous malformation].
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A 32-year-old right-handed female tour guide developed a transient prosopagnosia after the total removal of an arteriovenous malformation in the right occipital lobe. Neurological examination revealed only a left homonymous hemianopsia. The ablation on the right side involved total occipital lobe,
[Aphasia, prosopagnosia and mania: a case diagnosed with right temporal variant semantic dementia].
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Neurologic disorders can produce "secondary" mania, and clinicians must distinguish secondary mania from bipolar disorders (BD). Patients with new and late onset mania require an evaluation that includes a thorough history, a neurologic examination, neuroimaging, and other selected tests. Neurologic
Prosopagnosia Induced by a Left Anterior Temporal lobectomy Following a Right Temporo-occipital Resection in a Multicentric Diffuse Low-Grade Glioma.
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BACKGROUND
Face recognition is a complex function sustained by a distributed large-scale neural network, with a core system involving the ventral occipitotemporal cortex, the inferior longitudinal fasciculus (ILF), and the splenial commissural fibers. This circuit seems to be bilaterally organized,
Seeing but not recognizing.
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Visual recognition disturbances are caused by lesions that affect visual cortex as well as white matter connections between visual cortex and temporal and parietal cortex. Homonymous visual field defects are often present but do not explain the recognition difficulty. In "alexia without agraphia"
[Higher visual disorders].
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Higher visual disorders as discussed in this paper comprise visual hallucinations, palinopsy, hemineglect, Balint Holmes syndrome, prosopagnosia, visual objectagnosia, alexia without agraphia and cerebral achromatopsia. Such disorders are frequently caused by ischemia, but tumors, trauma and
Acquired cerebral dyschromatopsia.
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Color blindness developed in five patients apparently because of lesions in the posterior portions of both cerebral hemispheres. Three of them also had symptoms of prosopagnosia. The lesions were neoplastic in two and vascular in three of the patients. It would appear that bilateral, inferior,
Characteristics in limbic encephalitis with anti-adenylate kinase 5 autoantibodies.
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To report 10 patients with limbic encephalitis (LE) and adenylate kinase 5 autoantibodies (AK5-Abs).
We conducted a retrospective study in a cohort of 50 patients with LE with uncharacterized autoantibodies and identified a specific target using immunohistochemistry, Western blotting,
Non-invasive Mapping of Face Processing by Navigated Transcranial Magnetic Stimulation.
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Background: Besides motor and language function, tumor resections within the frontal and parietal lobe have also been reported to cause neuropsychological impairment like prosopagnosia. Objective: Since non-navigated transcranial magnetic stimulation (TMS) has previously been used to map
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