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respiratory insufficiency/sembap
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Management of acute respiratory failure due to pulmonary edema with nasal positive pressure support.
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The management of patients with respiratory failure from cardiogenic pulmonary edema may require intubation and mechanical ventilation. This provides both ventilatory assistance as well as the beneficial hemodynamic effects of positive intrathoracic pressure. As the need for ventilation is usually
Non-invasive pressure support ventilation in patients with respiratory failure due to severe acute cardiogenic pulmonary edema.
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BACKGROUND
Recent studies suggest the use of non-invasive pressure support ventilation (NIPSV) in patients with acute cardiogenic pulmonary edema (ACPE). However, it remains unclear whether all patients with ACPE benefit from NIPSV.
OBJECTIVE
To investigate short-term effects of NIPSV on
Pulmonary edema: pathophysiology, methods of measurement, and clinical importance in acute respiratory failure.
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Pulmonary edema is a common component of adult respiratory failure. The edema process is much more complex than simply excess water and/or protein crossing the microvascular membrane. The interstitial space itself is an active component of pulmonary edema, as are the alveolar type I and type II
High-Flow Nasal Cannula in Early Emergency Department Management of Acute Hypercapnic Respiratory Failure Due to Cardiogenic Pulmonary Edema.
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BACKGROUND
Noninvasive ventilation (NIV) is the recommended ventilatory support for acute cardiogenic pulmonary edema (CPE) associated with acute respiratory failure or hypercapnia. High-flow nasal cannula (HFNC) has emerged as an alternative to NIV in acute hypoxemic respiratoryPulmonary edema and hemorrhage, possible causes of pulmonary infection and respiratory failure in the early stage of lower spinal cord injury.
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Pulmonary infection and respiratory failure are frequently encountered in the early stage of acute spinal cord injury (SCI) and are thought of as the chief causes of death. Unfortunately, there is little knowledge concerned with the pathogenesis of pulmonary infection, respiratory failure and other
Pulmonary edema and respiratory insufficiency in acute pancreatitis.
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Pulmonary edema, cardiac enlargement, and respiratory insufficiency may occur in patients with acute pancreatitis. The mechanisms are complex and incompletely understood, but probable etiologic factors include fluid overload, left ventricular failure, impaired respiratory excursion and
Pulmonary edema and acute hypercapnic respiratory failure treated with bi-level nasal-CPAP: case report.
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Noninvasive mechanical ventilation has been suggested for the treatment of patients with respiratory failure. We describe the case of a patient affected by bilateral cystic bronchiectasis and acute hypercapnic respiratory failure, due to a cardiogenic pulmonary edema, successfully treated with
[Acute decompensation in chronic obstructive respiratory insufficiency. Pathogenesis of edema and role of noninvasive mechanical ventilation].
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Chronic obstructive pulmonary disease (COPD) is rather common. It is nearly always associated with excessive smoking. In advanced stages of COPD, patients have severe obstruction of airways and develop often acute episodes of respiratory failure, commonly due to broncho-pulmonary infections. This
Treatment of Hypertensive Cardiogenic Edema with Intravenous High-Dose Nitroglycerin in a Patient Presenting with Signs of Respiratory Failure: A Case Report and Review of the Literature.
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BACKGROUND Pulmonary edema is the accumulation of fluid in the lung secondary to increased hydrostatic pressure. Hypertensive cardiogenic pulmonary edema presents with a sudden onset of severe dyspnea, tachycardia, and tachypnea, and can occur when the systolic blood pressure exceeds 160 mmHg in
Negative pressure pulmonary edema following naloxone administration in a patient with fentanyl-induced respiratory depression.
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Naloxone is commonly used to reverse narcotic intoxication. However, its use is not entirely free of hazards. For instance, pulmonary edema (PE) has been reported to arise with the mechanism of over-sympathetic discharge caused by release of cat-echolamine or central neurogenetic responses to
Respiratory failure without pulmonary edema following injection of a glutamate agonist into the ventral medullary raphe of the rat.
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Injection of ibotenic acid (IA), a glutamate agonist, into the ventral medullary raphe (VMR; especially the nucleus raphe magnus) of the rat produced respiratory failure and death following a predictable course of events. The response to the IA injection was characterized initially by increased
Relapsing polychondritis with initial presentations of recurrent negative-pressure pulmonary edema and acute respiratory failure.
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Relapsing polychondritis is a rare autoimmune disease causing inflammation in cartilaginous structures and other tissues throughout the body. Negative-pressure pulmonary edema (NPPE) due to laryngeal swelling from relapsing polychondritis is rare and has not been reported. Here, we report a case of
APACHE II score and mortality in respiratory failure due to cardiogenic pulmonary edema.
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We reviewed retrospectively 88 patients to assess whether the APACHE II severity of disease classification system can predict mortality in patients with respiratory failure due to cardiac pulmonary edema. Mean score for survivors was higher than for nonsurvivors (24.5 +/- 6.7 vs. 20.7 +/- 5.7, p
Mediastinal teratoma with hydrops fetalis in a newborn and development of chronic respiratory insufficiency.
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BACKGROUND
Mediastinal fetal teratoma can be detected as a mass in the chest during a routine prenatal ultra-sound screening. Because of the pressure on mediastinal structures it can be the cause of non-immune hydrops fetalis and polyhydramnion. The development of hydrops fetalis leads to fetal
Effects of non-invasive ventilation in patients with acute respiratory failure excluding post-extubation respiratory failure, cardiogenic pulmonary edema and exacerbation of COPD: a systematic review and meta-analysis.
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BACKGROUND
This meta-analysis compared the effects of non-invasive ventilation (NIV) with invasive mechanical ventilation (InMV) and standard oxygen (O2) therapy on mortality and rate of tracheal intubation in patients presenting acute respiratory failure (ARF).
METHODS
We searched the MEDLINE,
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