rhododendron/nekrosis

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Phytophthora kernoviae sp. nov., an invasive pathogen causing bleeding stem lesions on forest trees and foliar necrosis of ornamentals in the UK.

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A new Phytophthora pathogen of trees and shrubs, previously informally designated Phytophthora taxon C, is formally named here as P. kernoviae. P. kernoviae was discovered in late 2003 during surveys of woodlands in Cornwall, south-west England, for the presence of another invasive pathogen, P.

Two novel flavonoids from the leaves of Rhododendron dauricum L. with their inhibition of TNF-α production in LPS-induced RAW 264.7 cells.

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Two new flavonoids, (2S)-6,8-dimethyl-5,7,3',4'-tetrahydroxyflavanone 4'-O-β-D-glucopyranoside (1) and quercetin 3-O-β-D-(6''-p-methoxybenzoyl)-galactopyranoside (2), together with ten known flavonoids (3-12) were isolated from the leaves of

First Report of Phytophthora ramorum on Viburnum bodnantense in Belgium.

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In the past decade, a new Phytophthora species inducing shoot canker on Rhododendron and dieback of Viburnum has been observed in Europe, mainly in Germany and the Netherlands, and California. This new pathogen has been named Phytophthora ramorum (3). In May 2002, a diseased Viburnum plant (Viburnum

Foliar Blight and Shoot Dieback Caused by Phytophthora ramorum on Viburnum tinus in the Pistoia Area, Tuscany, Central Italy.

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In spring 2013, pot-grown Viburnum tinus plants shipped to an ornamental nursery in Pescia (Pistoia, central Italy, 287 m a.s.l., 43°54'0″ N, 10°41'0″ E) from another local nursery were found to bear disease symptoms. Symptoms included brown to black foliar lesions, later expanding into larger

Rhododendrin ameliorates skin inflammation through inhibition of NF-κB, MAPK, and PI3K/Akt signaling.

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A wide range of active compounds isolated from nature is used in clinical applications and as a source of lead compounds for drug development. Rhododendron brachycarpum has been used as an oriental herbal medicine for skin inflammatory diseases. In this study, we isolated rhododendrin from

Immunomodulatory effects of epicatechin-(2β→O→7, 4β→8)-ent-epicatechin isolated from Rhododendron spiciferum in vitro.

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BACKGROUND Many traditional Chinese medicines (TCMs) can act as either immunosuppresants or immunostimulants, properties that have lead to their increasing use as immunomodulators in the treatment of disease. Recently, our lab successfully identified a dimer epicatechin-(2β→O → 7,

Anti-inflammatory effects of hyperoside in human endothelial cells and in mice.

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High-mobility group box 1 (HMGB1) was recently shown to be an important extracellular mediator of systemic inflammation, and endothelial cell protein C receptor (EPCR) has been shown to be involved in vascular inflammation. Hyperoside is an active compound isolated from Rhododendron brachycarpum G.

Phytophthora tropicalis Isolated from Diseased Leaves of Pieris japonica and Rhododendron catawbiense and Found in Irrigation Water and Soil in Virginia.

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An unidentified species of Phytophthora was isolated from irrigation water at a production nursery in Suffolk, VA in 2000 and 2001. Water samples were assayed using a filtration method (3). A similar species was recovered from soil samples collected in two mixed-hardwood forests in Fairfax County in

Rhododendron album Blume inhibits iNOS and COX-2 expression in LPS-stimulated RAW264.7 cells through the downregulation of NF-κB signaling.

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Rhododendron album Blume (RA) has traditionally been used as an herbal medicine and is considered to have anti-inflammatory properties. In the present study, we screened RA extracts with anti-inflammatory properties. The biological effects of an RA methanol extract (RAME) on inflammation were

Rhododendron album Blume extract inhibits TNF-α/IFN-γ-induced chemokine production via blockade of NF-κB and JAK/STAT activation in human epidermal keratinocytes.

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Rhododendron album Blume (RA) has traditionally been used as an herbal medicine and is considered to have anti‑inflammatory properties. It is a well‑known medicine for treatment of allergic or atopic diseases. In the present study, the biological effects of an RA methanol extract (RAME) on

The anti-inflammatory properties of Rhododendron molle G. Don leaf extract in LPS-induced RAW 264.7

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Rhododendron molle G. Don is a well-known traditional medicine which has been used to treat rheumatic inflammation. In this study, an inflammatory model of lipopolysaccharide (LPS)-stimulated RAW264.7 cells was established to analyze the anti-inflammatory effect and potential mechanism of the

Detection of Phytophthora ramorum Blight in Oregon Nurseries and Completion of Koch's Postulates on Pieris, Rhododendron, Viburnum, and Camellia.

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Phytophthora ramorum, the cause of sudden oak death in California and Oregon coastal forests and ramorum blight in European nurseries and landscapes (1), was detected in six Oregon nurseries in Jackson, Clackamas, and Washington counties from May to June 2003. The pathogen was isolated from:

First Report of Pycnostysanus azaleae on Rhododendron in Italy.

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Rhododendron species cultivation has a long history in northern Italy, where a wide selection of cultivars and hybrids is grown. In spring 2001, a previously unknown bud blast was observed on several rhododendron cultivars growing in gardens and parks in the Province of Biella, Italy. Flower

First Report of Phytophthora ramorum on Rhododendron in Greece.

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In April 2010, during a survey conducted in Fthiotis Prefecture of central Greece, symptoms of stem necrosis and leaf lesions were observed on two container-grown plants of Rhododendron, hybrid 'Kate Waterer' in a nursery. From symptomatic leaves, a Phytophthora species was isolated on PARPH-V8

Biological control of Phytophthora ramorumon rhododendron.

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Phytophthora ramorum was found in Poland in 2000 as the causal agent of rhododendron blight. Besides eradication of diseased plants and rhododendron growing around, chemical and biological control of the pathogen is necessary. In this study in vitro activity of grapefruit extract and chitosan in the

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