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serum sickness/protease
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Removal of glomerular immune complexes in passive serum sickness nephritis by treatment in vivo with proteolytic enzymes.
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The effect of in vivo protease treatment on glomerular immune complex deposits was investigated in passive serum sickness in mice and rats. BALB/c mice were injected intravenously with soluble immune complexes (IC) of cationic bovine gamma-globulin (BGG) antigen and rabbit anti-BGG antibody, or IC
Human monoclonal ScFv that inhibits cellular entry and metalloprotease activity of tetanus neurotoxin.
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Tetanus is a deadly disease of warm blooded animals and humans caused by an exotoxin called tetanospasmin or tetanus neurotoxin (TeNT) produced by anaerobic bacterium named Clostridium tetani TeNT is an A-B toxin; each molecule consists of a heavy chain (HC) containing cellular receptor binding
Coagulopathy after snake bite by Bothrops neuwiedi: case report and results of in vitro experiments.
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Coagulation studies were performed in a patient who had been bitten by a snake of the species Bothrops neuwiedi. The patient presented with hemorrhagic necrosis at the envenomization site and considerable bleeding from venous puncture sites. He developed a severe defibrination syndrome with a
Clinical pharmacokinetics and use of infliximab.
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Tumor necrosis factor-alpha (TNFalpha) is a key proinflammatory cytokine involved in chronic inflammatory diseases. Infliximab, a chimeric (human-murine) monoclonal IgG1 anti-TNFalpha antibody, is used in the treatment of Crohn's disease (including fistulising disease) and rheumatoid arthritis (in
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