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acetylstrophanthidin/hypoxia

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GreinarKlínískar rannsóknirEinkaleyfi
3 niðurstöður

Effect of acute hypoxia and hypercapnic acidosis on the development of acetylstrophanthidin-induced arrhythmias.

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The effect of acutely induced hypoxia, hypercapnic acidosis, and the combination of the two on the amount of acetylstrophanthidin (AS) required to produce cardiac arrhythmias was determined in anesthetized dogs. Each animal was studied during ventilation with room air and again during ventilation
The aim of this study was to determine if "ischemia" and/or reperfusion potentiate digitalis toxicity through effects on oscillatory afterpotentials. Isolated canine Purkinje tissue-papillary muscle preparations were studied using standard microelectrode techniques. Tissues were superfused for 10

Electrophysiological effects of insulin on normal and depressed cardiac tissues.

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The electrophysiological effects of insulin were studied in canine false tendons (FT) and papillary muscles from kittens and rats. Insulin caused hyperpolarization (2-8 mV) and an increase in action potential (AP) amplitude. However, the rate of rise and duration of the AP, the slope of phase 4
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