alpha 1-antitrypsin deficiency/glutathione

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GreinarKlínískar rannsóknirEinkaleyfi

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Decreased glutathione and low catalase activity contribute to oxidative stress in children with α-1 antitrypsin deficiency.

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BACKGROUND Recent investigations in animal models have revealed oxidative stress and oxidative damage in the pathogenesis of alpha-1 antitrypsin deficiency (AATD). However, no data are available on the oxidative stress status and antioxidant enzyme activity in these patients. This study was aimed to

Oxidative stress contributes to liver damage in a murine model of alpha-1-antitrypsin deficiency.

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Alpha-1-antitrypsin deficiency is a genetic disorder resulting in the expression of misfolded mutant protein that can polymerize and accumulate in hepatocytes, leading to liver disease in some individuals. Transgenic PiZ mice are a well-characterized model, which express human alpha-1-antitrypsin

Is there a therapeutic role for selenium in alpha-1 antitrypsin deficiency?

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Selenium is an essential trace mineral of fundamental importance to human health. Much of its beneficial influence is attributed to its presence within selenoproteins, a group of proteins containing the rare amino acid selenocysteine. There are 25 known human selenoproteins including glutathione

Alpha-1 antitrypsin binds preprohepcidin intracellularly and prohepcidin in the serum.

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Recent discoveries have indicated that the hormone hepcidin plays a major role in the control of iron homeostasis. Hepcidin regulates the iron level in the blood through the interaction with ferroportin, an iron exporter molecule, causing its internalization and degradation. As a result, hepcidin

Aerosolized Medications for Gene and Peptide Therapy.

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Inhalation therapy has matured to include drugs that: (1) deliver nucleic acids that either lead to the restoration of a gene construct or protein coding sequence in a population of cells or suppress or disrupt production of an abnormal gene product (gene therapy); (2) deliver peptides that target

[Genetic risk factors for chronic obstructive pulmonary disease (COPD)].

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Cigarette smoking has been shown to be a major environmental risk factor predisposing to the development of chronic obstructive pulmonary disease (COPD). However, only 10-20% of cigarette smokers develop COPD, implying undue susceptibility compared with the remainder of the population at large.

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Herbal & Natural Medicine

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