asphyxia neonatorum/phosphatase

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Serum alkaline phosphatase in pregnancy. II. Serial HSAP65degreesC estimations in pregnancy complicated with hypertension and pre-eclampsia.

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This is a study of heat-stable alkaline phosphatase (HSAP65degreesC) concentrations in the serum of pregnant women with hypertension (42 cases), mild preeclampsia (40 cases) and severe pre-eclampsia (22 cases). The results are seen in relationship to the occurrence of intrauterine fetal death,

Liver dysfunction in severe birth asphyxia.

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A total of 46 full term, appropriate for gestational age neonates were included in this study. Out of this, 31 infants had suffered severe birth asphyxia, while 15 normal babies formed the control group. The liver function tests of the asphyxiated group was deranged in 64.52% babies. There was 60%

Status of liver enzymes in babies with perinatal asphyxia.

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Because of hypoxemia, different organ systems of the body are affected in perinatal asphyxia. In this study, the functional status of liver of the asphyxiated babies was assessed through estimation of liver enzymes to see any correlation existing between enzyme changes and severity of perinatal

Fetal asphyxia induces acute and persisting changes in the ceramide metabolism in rat brain.

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Fetal asphyctic preconditioning, induced by a brief episode of experimental hypoxia-ischemia, offers neuroprotection to a subsequent more severe asphyctic insult at birth. Extensive cell stress and apoptosis are important contributing factors of damage in the asphyctic neonatal brain. Because

A microRNA-152 that targets the phosphatase and tensin homolog to inhibit low oxygen induced-apoptosis in human brain microvascular endothelial cells.

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Brain damage caused by perinatal asphyxia is dangerous for neonatal infants, but the mechanism by which it occurs remains elusive. In this study, microRNA-152 (miR-152) expression was induced by low oxygen levels in rat models of hypoxia brain damage, as well as in human brain microvascular

[Clinical significance of serum AKP, HSAP, LAP and gamma-GT determination in high-risk pregnancy].

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Serum levels of alkaline phosphatase (AKP), heat stable alkaline phosphatase (HSAP), leucine aminopeptidase (LAP) and gamma-glutamyl transferase(gamma-GT) were determined in 249 blood samples from 202 women with normal or high-risk pregnancy at 30-42 weeks. The results showed that AKP and HSAP

Changes in lysosomal hydrolases from cord blood of vigorous and asphyxiated Nigerian newborn infants.

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The activities of lysosomal hydrolases, beta-glucuronidase and acid phosphatase, were assayed in cord sera from forty-seven non asphyxiated babies and eleven babies who were severely asphyxiated at birth. Significant changes of these enzymes were observed in cord serum. Compared to the control

Hepatic dysfunction in asphyxiated neonates: prospective case-controlled study.

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OBJECTIVE This study was performed to determine the occurrence of hypoxic hepatitis in full-term neonates after perinatal asphyxia and to correlate between the rise in enzymes and severity of asphyxia with Apgar score and hypoxic ischemic encephalopathy (HIE) grading of the neonates. METHODS This

Enhancement of autophagic flux after neonatal cerebral hypoxia-ischemia and its region-specific relationship to apoptotic mechanisms.

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The multiplicity of cell death mechanisms induced by neonatal hypoxia-ischemia makes neuroprotective treatment against neonatal asphyxia more difficult to achieve. Whereas the roles of apoptosis and necrosis in such conditions have been studied intensively, the implication of autophagic cell death

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