cotinine/heilablóðfall

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Passive smoking and risk of coronary heart disease and stroke: prospective study with cotinine measurement.

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OBJECTIVE To examine the associations between a biomarker of overall passive exposure to tobacco smoke (serum cotinine concentration) and risk of coronary heart disease and stroke. METHODS Prospective population based study in general practice (the British regional heart study). METHODS 4729 men in

Association between serum cotinine levels and electrocardiographic left atrial abnormality.

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BACKGROUND Although the harmful effect of tobacco exposure on cardiovascular disease (CVD) and its risk factors are well established, the constituents of cigarette-smoke and the pathophysiological mechanism involved are unknown. Recently, deep terminal negativity of P wave in V1 (DTNPV1) has emerged

Protein kinase C family members as a target for regulation of blood-brain barrier Na,K,2Cl-cotransporter during in vitro stroke conditions and nicotine exposure.

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OBJECTIVE The aim of the study is to identify specific protein kinase C (PKC) isoforms involvement in K(+) transport mediated at altered blood-brain barrier (BBB) response to stroke conditions with prior nicotine exposure, which provides ways to intervene pharmacologically in PKC-mediated molecular

Association of Secondhand Smoke With Stroke Outcomes.

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Approximately half of never smokers are exposed to secondhand smoke (SHS). Smoking is a well-established stroke risk factor, yet associations between SHS, stroke, and poststroke mortality remain uncertain. We aimed to determine the prevalence of exposure to SHS among those with and without stroke

Cotinine-assessed second-hand smoke exposure and risk of cardiovascular disease in older adults.

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OBJECTIVE To examine whether second-hand smoke (SHS) exposure measured by serum cotinine is associated with increased coronary heart disease (CHD) and stroke risk among contemporary older British adults. METHODS Prospective population-based study with self-reported medical history and health

Nicotine and electronic cigarette (E-Cig) exposure decreases brain glucose utilization in ischemic stroke.

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Previous studies in our laboratory have shown that nicotine exposure decreases glucose transport across the blood-brain barrier in ischemia-reperfusion conditions. We hypothesize that nicotine can also dysregulate brain parenchymal glucose utilization by altering glucose transporters with effects on

Nicotine and cotinine modulate cerebral microvascular permeability and protein expression of ZO-1 through nicotinic acetylcholine receptors expressed on brain endothelial cells.

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The blood-brain barrier (BBB) adapts to a variety of pathological processes. Little is known about the effects of nicotine exposure on BBB function and the ability to adapt to stroke conditions. We have demonstrated, using a well-characterized in vitro BBB model, bovine brain microvessel endothelial

Regulation of blood-brain barrier Na,K,2Cl-cotransporter through phosphorylation during in vitro stroke conditions and nicotine exposure.

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Nicotine, a major constituent of tobacco smoke, has important effects on brain recovery after focal ischemia (Wang et al., 1997). The purpose of this work is to systematically test the effects of nicotine during stroke conditions on blood-brain barrier (BBB) potassium transport, protein expression

[Development and evaluation of a web-based education program to prevent secondary stroke].

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OBJECTIVE This study was conducted to develop and evaluate a web-based education program for secondary stroke prevention. METHODS A web-based secondary stroke prevention education program was developed using the system's life cycle methods and evaluated by comparing the effects of education among

Trends in Active Cigarette Smoking Among Stroke Survivors in the United States, 1999 to 2018.

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Background and Purpose- Patients who continue to smoke after a stroke face a higher risk of recurrent stroke. While several effective drugs for smoking cessation became available over the past 2 decades, whether active smoking has decreased among stroke survivors is unknown. We, therefore, evaluated

Symptoms of depression and active smoking among survivors of stroke and myocardial infarction: An NHANES analysis

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Historic concerns about the cardiovascular and neuropsychiatric side effects of smoking-cessation pharmacotherapy have in part limited their use. We sought to evaluate whether depressive symptoms are associated with active smoking among survivors of stroke and myocardial infarction (MI). To do this,

Impact of polysubstance use on high-sensitivity cardiac troponin I over time in homeless and unstably housed women

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Introduction: The use of controlled substances like cocaine increases the risk of cardiovascular disease (CVD) and myocardial infarction (MI). However, outside of alcohol and tobacco, substance use is not included in CVD risk assessment

Nicotine induces tissue factor expression in cultured endothelial and smooth muscle cells.

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OBJECTIVE Cigarette smoking is associated with an increased risk to develop myocardial infarction and ischemic stroke. However, the mechanisms responsible for these effects are still poorly understood. OBJECTIVE To investigate whether nicotine, the major component of cigarette smoking, and its main

Secondhand smoke exposure and intermittent claudication: a Scotland-wide study of 4231 non-smokers.

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OBJECTIVE Active smoking is an important risk factor for peripheral arterial disease. In contrast, published evidence on the association with secondhand smoke (SHS) is very limited. The aim of this study was to examine the association between SHS exposure and intermittent claudication (IC) among

The effect of hidden female smoking on the relationship between smoking and cardiovascular disease.

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Smoking is a known risk factor for cardiovascular morbidity and mortality, but several Korean studies have shown differing results on the association of current smoking status and the risk of cardiovascular disease (CVD). The aim of the present study was to investigate the association

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