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hypercapnia/blæðing

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Subarachnoid hemorrhage following permissive hypercapnia in a patient with severe acute asthma.

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In this article, we describe a case of a subarachnoid hemorrhage (SAH) in an acute severe asthma patient following mechanical hypoventilation. A 49-year-old man was admitted to an Intensive Care Unit with an acute exacerbation of asthma. After 3 days of mechanical ventilation (hypercapnia and

Subarachnoid hemorrhage in a child with status asthmaticus: significance of permissive hypercapnia.

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OBJECTIVE To report an 11-yr-old boy with acute status asthmaticus being managed with permissive hypercapnia who developed a subarachnoid hemorrhage during the course of his illness. METHODS An eight-bed pediatric intensive care unit in a community hospital. METHODS The patient was intubated for

Quantitative measurement of blood flow velocity in feline pial arteries during hemorrhagic hypotension and hypercapnia.

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Due to methodologic difficulties, few investigations have been made on the blood flow velocity in the cerebral microcirculation. Using a newly developed video camera method, we simultaneously measured the blood flow velocity and diameter of pial arteries during hemorrhagic hypotension, after blood

Hypercapnia potentiates renal vasoconstriction during hemorrhagic hypotension in awake rabbits.

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Although both hemorrhagic hypotension and hypercapnia increase renal vascular resistance (RVR) modestly, effects of interaction between these stimuli on RVR have not been examined systematically in unanesthetized animals. The purpose of this study was to test the hypothesis that renal

Local variations in the cerebral microcirculatory response to hypercapnia and haemorrhage.

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This study evaluates local variations of the cerebral vasomotor responses to hypercapnia and haemorrhagic hypotension in a pig model. Four laser Doppler flow probes were used in each pig. There was considerable variation in laser Doppler signals between the four probes in baseline recordings. The
OBJECTIVE Lung protective ventilation has a beneficial effect in treating patients with acute respiratory distress syndrome (ARDS). An effect of this ventilation modality is hypercapnia, which leads to increased cerebral blood flow. Since increased cerebral blood flow can induce brain oedema the
BACKGROUND Anesthetic management during functional endoscopic sinus surgery is aimed at minimizing bleeding and establishing a near-perfect surgical field. We investigated whether deliberate intraoperative hypercapnia and hypocapnia may affect blood loss and quality of surgical field through a
To examine the effects of subarachnoid tetracaine and epinephrine on spinal cord blood flow (SCBF), lumbar SCBF and cerebral blood flow (CBF) were measured simultaneously by the hydrogen clearance technique in dogs (n = 45) anesthetized with halothane. The lumbar subarachnoid administration of

Humoral response and blood pressure regulation during hypercapnia and haemorrhage in dogs.

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1. The blood-bathed organ technique was used to study the release of catecholamines, angiotensin II and prostaglandin-like (PL) substances into the circulation during hypercapnia and after haemorrhage in anaesthetized dogs. 2. Elevated blood concentrations of noradrenaline, angiotensin II and

Alpha-adrenoreceptor antagonists and pial vessel diameter during hypercapnia and hemorrhagic hypotension in the cat.

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It has been proposed that sympathetic activation may prevent maximum dilatation of extra-parenchymal cerebral vessels during hemorrhagic hypotension and hypercapnia. In the present study, the effect of alpha-adrenoreceptor antagonists (phenoxybenzamine 1.5 mg kg-1 or phentolamine 8 mg kg-1) on pial
Aim: Safe limits of arterial partial pressure of carbon dioxide (PaCO2) and acidosis in premature infants are not well defined. Both respiratory and systemic illness along with center-specific ventilation strategies contribute to PaCO2 fluctuations and acid-base
1. Late cerebral arterial spasm was induced by repeated injections of autologous blood in a total amount of 14-33 ml into the basal cisterns of baboons to mimick subarachnoid hemorrhage (SAH). Regional cerebral blood flow (CBF), sagittal sinus pressure, cerebral arterial caliber from angiograms, and

Acidemia versus hypercapnia and risk for severe intraventricular hemorrhage.

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In extremely low birth weight (ELBW) infants, levels of hypercapnia (Paco 2) > 60 mm Hg are considered a risk factor for severe intraventricular hemorrhage (IVH). Since cerebral vasoreactivity depends on arterial pH (apH) rather than Paco 2, we hypothesize that the role of mild-to-moderate
OBJECTIVE To examine whether hypercapnia in very low birth weight (VLBW) infants during the first 3 days of life is associated with severe intraventricular hemorrhage (IVH). METHODS Retrospective cohort study of inborn VLBW infants between January 1999 and May 2004 with arterial access during the

Pial vessel caliber and cerebral blood flow during hemorrhage and hypercapnia in the rabbit.

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We examined the relationship between cerebral blood flow (CBF) and pial vessel caliber responses to graded hemorrhagic hypotension at both normocapnia and hypercapnia in 31 anesthetized rabbits. Changes in CBF (hydrogen clearance) and pial arteriolar diameter (image splitting) were predictably
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