hypercapnia/seizures

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Seizure susceptibility during recovery from hypercapnia in neonatal dogs.

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Seizure susceptibility during recovery from hypercapnia was investigated in seven anesthetized neonatal dogs; 13, 20, or 30% CO2 gas was administered for 30 min through a ventilator to result in three levels of hypercapnia in which measured PaCO2 values were approximately 70, 100, and 140 mm Hg.

Sensitivity to seizure-like activity in Drosophila following acute hypoxia and hypercapnia.

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Human seizure disorders represent a heterogeneous collection of neuropathies, many of which are poorly understood. To investigate the etiology of seizure disorders, we have used a group of Drosophila mutants known as the bang-sensitive (BS) paralytics. The BS mutants exhibit seizure-like activity

A novel approach for selective brain cooling: implications for hypercapnia and seizure activity.

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OBJECTIVE During selective brain cooling (SBC) the brain temperature (TB) is reduced while the core temperature (TC) remains unchanged. This animal study investigated changes in brain temperature induced by a novel approach of cooling the brain from the pharynx (pSBC) and whether these temperature

Carbon monoxide regulates cerebral blood flow in epileptic seizures but not in hypercapnia.

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Carbon monoxide (CO) is an endogenously produced gas sharing many properties with nitric oxide (NO), notably activating soluble guanylate cyclase and relaxing blood vessels. The brain can generate high quantities of CO from a constitutive enzyme, haem oxygenase (HO-2). To determine whether CO is

Hypercapnia-induced brain acidosis: Effects and putative mechanisms on acute kainate induced seizures.

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OBJECTIVE This study investigated the mechanism of the anticonvulsant effects of carbogen containing 5% CO2 in a kainic acid (KA) rat model. METHODS Four-week-old Sprague-Dawley rats were divided into four groups: control, carbogen, KA+air, and KA+carbogen. Carbogen containing 5% CO2 was applied

Respiratory changes with seizures in localization-related epilepsy: analysis of periictal hypercapnia and airflow patterns.

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OBJECTIVE The rate of sudden unexpected death in epilepsy (SUDEP) approaches 9 per 1,000 patient-years in patients with refractory epilepsy. Respiratory causes are implicated in SUDEP. We reported that ictal hypoxemia occurs in one-third of seizures in localization-related epilepsy. We now report on

The effect of hypercapnia and hypoxia on electrically induced convulsions in rabbits.

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[Hemodynamically-induced transitory ischemic attacks. A differential focal motor seizures diagnosis?].

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This case report intends to focus attention on hemodynamic TIAs as cause of repetitive involuntary movements (RIMs) as differential diagnosis of simple partial motor seizures. We report two patients with episodic unilateral limb shaking lasting up to several minutes and which could be triggered by

Glottal patency during experimental cortical seizures in piglets.

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OBJECTIVE Systemically-induced seizures produce glottal airflow obstruction in anesthetized pigs, resulting in hypercapnia and respiratory acidosis. Cortically-induced seizures may be more representative of human seizure disorders. The purpose of this study was to describe glottal area patency (GAP)

Cerebral metabolic responses to electroconvulsive shock and their modification by hypercapnia.

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Brain glucose metabolism was studied in paralyzed, ventilated rats given electroconvulsive shock (ECS) under normocapnic and hypercapnic conditions. Brains were obtained with a freeze-blowing apparatus. Rates of glucose utilization were determined with [2-14C]glucose and [3H]deoxyglucose as tracers.

Singultus, paper-bag ventilation, and hypercapnia.

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Sir Louis Francis Knuthsen (1869-1957), the physician who painstakingly listed almost all treatments known for obstinate hiccough, ascribes the holding of breath method to Philip Henry Pye-Smith, FRS (1840-1914), consultant at Guy's Hospital in London. In fact, the strategy is much older and was

Interacting effects of hypoxia adaptation and acute hypercapnia on oxygen tolerance in rats.

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To define the effects of hypoxia adaptation on tolerance to a useful range of O2 pressures, groups of 20 or more rats were exposed to O2 at 1.0, 1.5, 2.0, 3.0, and 4.0 ATA before and after adaptation to an inspired PO2 (PIO2) of 71 Torr for 5 days. Effects of acute hypercapnia on O2 tolerance in

Sudden infant death syndrome, sleep, and seizures.

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OBJECTIVE benign febrile seizures seen in 7% of infants before 6 months play a role in the terminal pathway in a subset of sudden infant death syndrome victims. Supporting evidence: (1) lack of 5-hydroxitryptamine, one consistent finding in sudden infant death syndrome that Kinney et al coined a

Endogenous heme oxygenase prevents impairment of cerebral vascular functions caused by seizures.

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In newborn pigs, the mechanism of seizure-induced cerebral hyperemia involves carbon monoxide (CO), the vasodilator product of heme catabolism by heme oxygenase (HO). We hypothesized that seizures cause cerebral vascular dysfunction when HO activity is inhibited. With the use of cranial window

Risk factors for oxygen toxicity seizures in hyperbaric oxygen therapy: case reports from multiple institutions.

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Oxygen toxicity seizures are a rare but recognized complication of hyperbaric oxygen (HBO2) therapy. Many patients undergoing HBO2 therapy have medical conditions or are taking medications that could contribute to seizures. Previous literature has not extensively reported on these factors in

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