hypernatremia/bjúgur

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[Edemas with hypernatremia. Mechanism and treatment (author's transl)].

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The discrepancy between the finding of hypernatremia--a manifestation of extra-cellular fluid depletion--and the presence of edema--a manifestation of increased ECF--is best explained by the patient's inability to mobilize edemas to compensate intra-vascular dehydratation. Treatment must start by

Hypernatremia with edema.

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Hypernatremia is usually associated with water depletion. Seven very ill patients developed hypernatremia in association with marked edema during therapy in the hospital. All patients had hypoalbuminemia and azotemia. At the time of hypernatremia, urine output averaged 1880 mL/24 h and urine sodium

Severe cerebral edema in a patient with anasarca and hypernatremia.

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We describe a woman whose fatal post-liver transplantation cerebral edema was unexpected and of unusual pathogenesis. Her severe cerebral edema is of considerable pathophysiologic interest: 1) it developed in the setting of marked anasarca and persistent hypernatremia, and 2) although hepatic

Induced and sustained hypernatremia for the prevention and treatment of cerebral edema following brain injury.

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Cerebral edema develops in response to and as a result of a variety of neurologic insults such as ischemic stroke, traumatic brain injury, and tumor. It deforms brain tissue, resulting in localized mass effect and increase in intracranial pressure (ICP) that are associated with a high rate of

Hypernatremia with edema.

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Fatal acute hypernatremia resulting from a massive intake of seasoning soy sauce

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Background: Hypernatremia due to salt poisoning is clinically rare and standard care procedures have not been established. We report a case of salt poisoning due to massive intake of seasoning soy sauce.

[Life-threatening hypernatremia caused by faulty preparation of fully adapted infant formula].
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Two male newborns developed severe life threatening hypernatremia with serum sodium levels of 181 and 196 meq/l respectively. Both children were fed a two-to fivefold concentrated powdered infant formula since birth. Shortly before admission diarrhea developed in both patients. The children were

Hypernatremia in dogs.
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Hypernatremia is potentially life threatening and is caused by imbalances in water and sodium that occur from either water loss or sodium gain, often in the presence of inadequate water intake. The resultant hyperosmolarity, if acute or severe, can result in rapid shifts of water from the

Hyponatremia and hypernatremia.
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The concentration of serum sodium is determined by the external balance of water. Hyponatremia occurs when total body water is in excess of sodium, and hypernatremia develops when body water is relatively decreased in relation to sodium. Both disorders may be present in patients with various disease

[Hypernatremia in head-injured patients: friend or foe?].
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Hypernatremia is defined by a serum sodium concentration of more than 145 mmol/L and reflects a disturbance of the regulation between water and sodium. The high incidence of hypernatremia in patients with severe brain injury is due various causes including poor thirst, diabetes insipidus, iatrogenic

Hypernatremia.
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Salt toxicity can be fatal in dogs and cats. Whether toxicity occurs accidentally or iatrogenically, it is important to recognize the clinical signs of sodium toxicosis, which are mainly caused by hypernatremia and associated cerebral edema. Treatment involves prompt initiation of fluid therapy

Hypernatremia in the elderly.
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Hypernatremia is common in elderly persons, who may present with signs that are easily overlooked. Prompt diagnosis and appropriate therapy can sometimes lead to a remarkable recovery. To avoid cerebral edema, rehydration must be accomplished gradually over 48 to 72 hours. The recommended

Hypernatremia with choroid plexus hematomas and elevated CSF protein level.
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A 3-month-old infant had hypernatremia, neurologic dysfunction, and an unusually high level of cerebrospinal fluid protein. The autopsy findings included cerebral edema, intravascular coagulation, and bilateral choroid plexus hematomas. Extensive destruction of the choroid plexus, in combination

Hypernatremia is associated with poorer outcomes following aneurysmal subarachnoid hemorrhage: a nationwide inpatient sample analysis.
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Hypernatremia is one of the most common electrolyte disturbances following aneurysmal subarachnoid hemorrhage (aSAH) and has been correlated with increased mortality in single institution studies. We investigated this association using a large nationwide healthcare

Effect of Hypernatremia on Outcomes After severe Traumatic Brain Injury: A Nationwide Inpatient Sample analysis.
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OBJECTIVE Induced hypernatremia is frequently used to reduce intracranial pressure in patients with severe traumatic brain injury (TBI). This technique is controversial, and some studies have independently associated hypernatremia with worse outcomes after TBI. We sought to investigate this

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hypernatremia/bjúgur

[Edemas with hypernatremia. Mechanism and treatment (author's transl)].

Hypernatremia with edema.

Severe cerebral edema in a patient with anasarca and hypernatremia.

Induced and sustained hypernatremia for the prevention and treatment of cerebral edema following brain injury.

Hypernatremia with edema.

Fatal acute hypernatremia resulting from a massive intake of seasoning soy sauce

[Life-threatening hypernatremia caused by faulty preparation of fully adapted infant formula].

Hypernatremia in dogs.

Hyponatremia and hypernatremia.

[Hypernatremia in head-injured patients: friend or foe?].

Hypernatremia.

Hypernatremia in the elderly.

Hypernatremia with choroid plexus hematomas and elevated CSF protein level.

Hypernatremia is associated with poorer outcomes following aneurysmal subarachnoid hemorrhage: a nationwide inpatient sample analysis.

Effect of Hypernatremia on Outcomes After severe Traumatic Brain Injury: A Nationwide Inpatient Sample analysis.

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