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hypertensive retinopathy/kalín

Krækjan er vistuð á klemmuspjaldið
GreinarKlínískar rannsóknirEinkaleyfi
3 niðurstöður

Protection of retinal vasculature by losartan against apoptosis and vasculopathy in rats with spontaneous hypertension.

Aðeins skráðir notendur geta þýtt greinar
Skráðu þig / skráðu þig
OBJECTIVE Retinal blood vessels may develop vasculopathy and apoptosis in response to hypertension. The present study was aimed at testing the role of losartan, a specific antagonist of angiotensin II receptor type 1 receptor in regulation of vascular apoptosis in retinal vasculature with

Apparent mineralocorticoid excess and the long term treatment of genetic hypertension.

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Apparent mineralocorticoid excess (AME) is a genetic disorder causing severe hypertension, hypokalemia, and hyporeninemic hypoaldosteronism owing to deficient 11 beta-hydroxysteroid dehydrogenase type-2 (11βHSD2) enzyme activity. The 11βHSD2 enzyme confers mineralocorticoid receptor specificity for

Hypertensive crisis with 2 target organ impairment induced by glycyrrhizin: A case report.

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BACKGROUND Glycyrrhizin is the main active component of licorice. Licorice and glycyrrhizin induced hypertension has been widely reported, yet licorice and glycyrrhizin induced hypertensive crisis has been rarely known. UNASSIGNED The case of this report was a 47-year-old woman, who took 225 mg of
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