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karwinskia/ganglion cysts

Krækjan er vistuð á klemmuspjaldið
GreinarKlínískar rannsóknirEinkaleyfi
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Depression of fast axonal transport produced by tullidora.

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The fast axoplasmic transport of labeled proteins was studied in cats showing hindlimb paralysis 4-7 weeks after a single oral dose of tullidora (Karwinskia humboldtiana) toxins. The isotope (3H-leucine) was injected into the spinal ganglion and the contralateral spinal cord of the seventh lumbar

Buckthorn neuropathy in vitro : evidence for a primary neuronal effect.

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The neuropathy produced by toxins isolated from the buckthorn shrub (Karwinskia humboldtiana) has previously been proposed as a model for primary PNS demyelination in vivo. These toxins have been suggested to act through a metabolic effect on the Schwann cell. The aim of the present study was to use

Depression of fast axonal transport in axons demyelinated by intraneural injection of a neurotoxin from K. humboldtiana.

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Tullidinol, a neurotoxin extracted from the Karwinskia humboldtiana fruit, dissolved in peanut oil was injected into the right sciatic nerve of adult cats. The contralateral sciatic nerve received an equivalent volume of peanut oil alone. The fast axonal transport of labeled ([3H]Leucine) protein
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