sickle cell trait/kalín

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Renal response to potassium loading in sickle cell trait.

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Anatomical and functional renal medullary abnormalities are common in patients with sickle cell disease (HbSS) and sickle cell trait (HbAS). Sickle cell disease is associated with impaired urinary potassium excretion, but renal potassium handling in patients with HbAS has not been reported. To

Effect of a 'sickling pulse' on calcium and potassium transport in sickle cell trait red cells.

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1. To trace the early development of the extensive functional membrane abnormalities found in sickle cell anaemia red cells which result from polymerization of haemoglobin S, we followed the effects on Ca and K transport of an in vitro sickling pulse in sickle cell trait (SA) red cells, whose

pH dependency of potassium efflux from sickled red cells.

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Potassium efflux from deoxygenated, hemoglobin S-containing red cells is often used as an "objective" in vitro measure of aed cell sickling, particularly during tests with antisickling agents. Since varying pH is known to affect both the extent of sickling and passive K+-flux across the red cell

Effects of gamma irradiation on red cells from donors with sickle cell trait.

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BACKGROUND Transfusion-associated graft-versus-host disease can be prevented by gamma irradiation of blood components. Red cells (RBCs) from sickle cell disease patients may exhibit oxidative changes of RBC membranes due to the instability of hemoglobin (Hb) S. Persons with sickle cell trait are

Sodium-potassium pump, ion fluxes, and cellular dehydration in sickle cell anemia.

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We studied the role of the sodium-potassium pump in erythrocytes of 12 patients with sickle cell anemia (SS). Ouabain-binding sites per cell and pump-mediated Rb/K uptake were significantly higher in SS patients than in white or black controls. Ouabain-resistant Rb/K influx was also greater than in

Storage related haematological and biochemical changes in Plasmodium falciparum infected and sickle cell trait donor blood.

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UNASSIGNED In sub-Saharan Africa where sickle cell trait (SCT) and malaria is prevalent, significant proportions of blood donors may be affected by one or more of these abnormalities. The haemato-biochemical properties of SCT and asymptomatic malaria in donor blood have not been evaluated. This

The altered autonomic nervous system activity in iron deficiency anemia.

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Autonomic function is impaired in anemic patients with various etiologies such as vitamin B12 deficiency, sickle cell trait, and thalassemia major. However, there are insufficient data about autonomic functions in patients with iron deficiency anemia, the leading cause for anemia in the general

Hyperkalemic hyperchloremic metabolic acidosis in sickle cell hemoglobinopathies.

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This report describes the occurrence of hyperkalemic hyperchloremic metabolic acidosis in six patients with sickle cell hemoglobinopathies. Three patients had sickle cell anemia, two had sickle cell trait and one had S-C disease. In all patients, decreased renal potassium excretion was demonstrated

Renal abnormalities in sickle cell disease.

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Sickle cell anemia and the related hemoglobinopathies are associated with a large spectrum of renal abnormalities. The patients have impaired urinary concentrating ability, defects in urinary acidification and potassium excretion, and supranormal proximal tubular function. The latter is manifest by

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