takotsubo cardiomyopathy/offita

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Does obesity affect the outcomes in takotsubo cardiomyopathy? Analysis of the Nationwide Inpatient Sample database, 2010-2014.

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BACKGROUND Obesity can lead to increased oxidative stress which is one of the proposed mechanisms in the etiopathogenesis of takotsubo cardiomyopathy (TCM). OBJECTIVE The presence of obesity adversely impacts clinical outcomes in TCM patients. METHODS We queried the Nationwide Inpatient Sample

A big man with a broken heart: stress-induced cardiomyopathy in a morbidly obese man.

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Etiologies, predictors, and economic impact of readmission within 1 month among patients with takotsubo cardiomyopathy.

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BACKGROUND Limited data exist on readmission among patients with takotsubo cardiomyopathy (TC), a commonly reversible cause of heart failure. OBJECTIVE We sought to identify etiologies and predictors for readmission among TC patients. METHODS We queried the National Readmissions Database for

Management of Takotsubo cardiomyopathy in non-academic hospitals in France: The Observational French SyndromEs of TakoTsubo (OFSETT) study.

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BACKGROUND Takotsubo cardiomyopathy (TTC) is a rare condition characterized by a sudden temporary weakening of the heart. TTC can mimic acute myocardial infarction and is associated with a minimal release of myocardial biomarkers in the absence of obstructive coronary artery disease. OBJECTIVE To

Takotsubo Cardiomyopathy and Chronic Kidney Disease: A Scoping Study.

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Sympathetic nervous system hyperactivity and elevated catecholamine levels are known features of chronic kidney disease (CKD). On the other hand, CKD itself is a high risk for Cardiovascular disease (CVD) and in fact most patients with CKD die before reaching dialysis. Furthermore, Many CKD risk

A case report of recurrent spontaneous coronary artery dissection and Takotsubo cardiomyopathy: a treatment dilemma.

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Spontaneous coronary artery dissection (SCAD) is an uncommon cause of acute coronary syndrome in younger females with no pre-existing history of coronary artery disease. Recurrent SCAD is common after a first episode and can involve the same coronary artery or present as a new

Metabolic stress-induced cardiomyopathy is caused by mitochondrial dysfunction due to attenuated Erk5 signaling.

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The prevalence of cardiomyopathy from metabolic stress has increased dramatically; however, its molecular mechanisms remain elusive. Here, we show that extracellular signal-regulated protein kinase 5 (Erk5) is lost in the hearts of obese/diabetic animal models and that cardiac-specific deletion of

Cyber war--when virtual fear of death turns into a real threat of life.

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Many electronic games have violent contents. A growing population of adolescent boys and girls report to regularly play violent electronic games (VEGs). Extensive video game use has been linked with obesity, physical discomfort and seizures. We report on a young, healthy man who participated in an

Reversible cardiomyopathies--a review.

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End-stage renal disease, cirrhosis, obesity, tachycardia, and extreme stress have all been shown to result in impaired left ventricular function. It is becoming clear, however, that the cardiomyopathies associated with these states are reversible after resolution of the underlying process. In this

Endoplasmic reticulum stress-induced iRhom2 up-regulation promotes macrophage-regulated cardiac inflammation and lipid deposition in high fat diet (HFD)-challenged mice: Intervention of fisetin and metformin.

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Endoplasmic reticulum stress (ERS) has been implicated in obesity-associated cardiac remodeling and dysfunction. Inactive rhomboid protein 2 (iRhom2), also known as Rhbdf2, is an inactive member of the rhomboid intramembrane proteinase family, playing an essential role in regulating inflammation.

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