theaflavin/atrophy

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Theaflavins prevent cartilage degeneration via AKT/FOXO3 signaling in vitro.

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Theaflavins (TFs) are the main bioactive polyphenols in tea and contribute to protection against oxidative stress. Excessive reactive oxygen species (ROS) accumulation can lead to the disruption of cartilage homeostasis. The present study examined the potential effects of TFs on H2O2‑induced

Theaflavins Improve Memory Impairment and Depression-Like Behavior by Regulating Microglial Activation.

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Inflammation in the brain is associated with various disorders including Alzheimer's disease and depression. Thus, inflammation has received increasing attention regarding preventive approaches to such disorders. Epidemiological investigations have reported that drinking tea reduces the risk of

Theaflavin ameliorates behavioral deficits, biochemical indices and monoamine transporters expression against subacute 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced mouse model of Parkinson's disease.

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Evidence from clinical and experimental studies indicates that degeneration of nigrostriatal dopaminergic neurons is a pathological hallmark of Parkinson's disease (PD). The present study was designed to investigate the neuroprotective potential of theaflavin (TF) on oxidative stress, monoamine

Theaflavin promotes myogenic differentiation by regulating the cell cycle and surface mechanical properties of C2C12 cells

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Aging and muscle diseases often lead to a decline in the differentiation capacity of myoblasts, which in turn results in the deterioration of skeletal muscles (SkM) function and impairment of regeneration ability after injury. Theaflavins, the "gold molecules" found in black tea, have been reported

Inhibitory effect on the apoptosis of disc nucleus pulposus cells in rats by silencing COL9A3 gene to mediate MAPK signaling pathway: a study on the function and mechanism

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Objective: To explore the effect of collagen IX alpha 3 chain (COL9A3) gene silencing on apoptosis of nucleus pulposus cells in rats with intervertebral disc degeneration (IVDD) and its regulatory mechanism, so as to provide potential

Tea polyphenols inhibit rat osteoclast formation and differentiation.

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Matrix metalloproteinases (MMPs) play an important role in degeneration of the matrix associated with bone and cartilage. Regulation of osteoclast activity is essential in the treatment of bone disease, including osteoporosis and rheumatoid arthritis. Polyphenols in green tea, particularly

Tea Polyphenols Inhibit Rat Osteoclast Formation and Differentiation.

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Neuroprotective Effects and Mechanisms of Tea Bioactive Components in Neurodegenerative Diseases.

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As the population ages, neurodegenerative diseases such as Parkinson's disease (PD) and Alzheimer's disease (AD) impose a heavy burden on society and families. The pathogeneses of PD and AD are complex. There are no radical cures for the diseases, and existing therapeutic agents for PD and AD have

Tea Polyphenols in Parkinson's Disease.

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Parkinson's disease (PD) is a common motor neurodegenerative disorder with multifactorial etiology that is an increasing burden on our aging society. PD is characterized by nigrostriatal degeneration which might involve oxidative stress, α-synuclein (αS) aggregation, dysregulation of redox metal

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