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anemia/antinfiammatorio
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Pagina 1 a partire dal 769 risultati
Induction of hemolytic anemia by nonsteroidal antiinflammatory drugs.
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The incidence of immune hemolytic anemia (IHA) is increasing. The proliferation of pharmaceuticals is a contributing factor to this increase. IHA is an uncommon, though significant, adverse effect of a wide variety of drugs. Several recent case reports have implicated the nonsteroidal
Low drug attributability of aplastic anemia in Thailand. The Aplastic Anemia Study Group.
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From 1989 to 1994, a population-based, case-control study of aplastic anemia was conducted in Thailand, including the regions of Bangkok, Khonkaen in the northeast, and Songkla in the south. An annual incidence in Bangkok of 3.7 cases per million population, about twice as high as in Western
Anemia in systemic lupus erythematosus.
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Anemia is a common finding in children with systemic lupus erythematosus (SLE). Anemia in SLE may result from several mechanisms and more than one may be operative at any time. Anemia of chronic disease is the most common, but a low hemoglobin can be caused by auto-antibodies to red cells as part of
[Drug-induced autoimmune hemolytic anemia].
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AUTOANTIBODY PRODUCTION: The production of autoantibodies can only occur if immune tolerance is circumvented. Thus drug-induced autoimmune hemolytic anemia requires that the drug have an effect on both autoantigens and on the immune system. AN EXAMPLE, METHYLDOPA: Methyldopa is a hypotensive agent
Sulindac-induced immune hemolytic anemia.
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BACKGROUND
Sulindac, a nonsteroidal, anti-inflammatory, indene-derived drug, caused life-threatening immune hemolytic anemia in an individual with back pain.
METHODS
A patient was admitted to the hospital with immune hemolytic anemia and kidney and liver failure after several days ingestion of
[The anti-inflammatory effect of hydrocortisone in experimental anemia].
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Anti-inflammatory effects of interleukin-35 in acquired aplastic anemia.
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Interleukin (IL)-35 is a novel regulatory cytokine primarily produced by regulatory T cells. Accumulating evidence has established that IL-35 plays an important role in the regulation of immune homeostasis, but little is known regarding the function of IL-35 in acquired aplastic anemia (AA). The aim
[Anemia in patients with rheumatoid arthritis].
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Anemia is characterized by a reduced count of red blood cells and/or low hemoglobin content in blood. The article reviews basic information on pathogenesis, differential diagnosis and treatment of anemia in patients with rheumatoid arthritis (RA). Anemia in this case is characterized as anemia of a
An approach to iron-deficiency anemia.
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Iron-deficiency anemia is a common reason for referral to a gastroenterologist. In adult men and postmenopausal women, gastrointestinal tract pathology is often the cause of iron-deficiency anemia, so patients are frequently referred for endoscopic evaluation. Endoscopy may be costly and at times
Immune hemolytic anemia associated with sulindac.
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A number of adverse reactions have been associated with sulindac, a nonsteroidal anti-inflammatory drug, but acute hemolytic anemia associated with sulindac has not yet been reported, to our knowledge. We encountered two cases of severe immune hemolytic anemia, one fatal, which appear to have been
Iron and anemia of chronic disease.
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Anemia of chronic disease (ACD) is the most frequent anemia found in hospitalized patients, often occurring in subjects suffering from chronic inflammatory disorders. The underlying diversion of iron traffic leads to a withdrawal of the metal from the sites of erythropoiesis and the circulation to
Epidemiology and aetiology of aplastic anemia.
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Epidemiological data show that the incidence of aplastic anemia is a least 4-5 times as high in the far East as in the West. The reason seems to be complex--a number of exogenic factors act against a background of genetic differences in susceptibility. A Swedish study of aplastic anemia shows the
Anemia, tumor hypoxemia, and the cancer patient.
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OBJECTIVE
To review the impact of anemia/tumor hypoxemia on the quality of life and survival in cancer patients, and to assess the problems associated with the correction of this difficulty.
METHODS
MEDLINE searches were performed to find relevant literature regarding anemia and/or tumor hypoxia in
[Oxidant-drug induced hemolytic anemia in dogs].
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Heinz body anemia was induced in dogs by consecutive oral administration of 200 mg/kg and 60 mg/kg of aminopyrine, 200 mg/kg of phenacetin and 5 mg/kg of acetylphenylhydrazine, for either 8 or 16 weeks. Biochemical analysis showed a decrease of haptoglobin in plasma and reduced-glutathion in red
Case report: hemolytic anemia produced by tolmetin.
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Tolmetin, a nonsteroidal anti-inflammatory drug, was found to be the etiologic agent in a case of drug-induced hemolytic anemia. A 35-year-old female who had ingested tolmetin sporadically in the past took two doses that resulted in acute hemolysis. Two days after taking the second dose, she had a
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