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azoospermia/potassio
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6 risultati
Gossypol-induced hypokalemia and role of exogenous potassium salt supplementation when used as an antispermatogenic agent in male langur monkey.
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In our earlier study, we have observed that hypokalemia in langur monkeys, following gossypol acetic acid (GAA) treatment (5 mg dose level) when used as an antispermatogenic agent, and potassium salt supplementation partially maintained body potassium level of the animals. The aims of the present
Sodium and potassium in normal and pathological seminal plasma.
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A total number of 273 seminal plasma samples from normozoospermia (n = 91), oligozoospermia (n. = 50), oligoasthenozoospermia (n = 76), asthenozoospermia (n = 10) and azoospermia (n = 46) were analysed for sodium and potassium. A decreased sodium level was seen in pathological groups. A negative
Sodium and potassium in normal and pathological seminal plasma.
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A total number of 273 seminal plasma samples from normozoospermia (n = 91), oligozoospermia (n = 50), oligo-asthenozoospermia (n = 76), asthenozoospermia (n = 10) and azoospermia (n = 46) were analysed for sodium and potassium. A decreased sodium level was seen in pathological groups. A negative
Prolactin injection, a new contraceptive method: experimental study.
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"Prolactin injection" is presented as a new contraceptive method. The method was tested in dogs. The dogs in the test group were injected with prolactin (PRL) in a dose of 600 micrograms/kg of body weight weekly for 6 months. During this period, the testicles, semen, reproductive hormones, renal
Antispermatogenic action of gossypol in men.
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Twelve Brazilian men were treated with gossypol for 1 year. The treatment schedule consisted of oral administration of 20 mg gossypol daily for 4 months, followed by a maintenance dose of 20 mg on alternate days (3 days a week), totaling 60 mg weekly for 8 months. Ten men became azoospermic at the
Mapping breakpoints of a familial chromosome insertion (18,7) (q22.1; q36.2q21.11) to DPP6 and CACNA2D1 genes in an azoospermic male.
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It is widely accepted that the incidence of chromosomal aberration is 10-15.2% in the azoospermic male; however, the exact genetic damages are currently unknown for more than 40% of azoospermia. To elucidate the causative gene defects, we used the next generation sequencing (NGS) to map the
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